What causes blepharospasms?

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From the Guidelines

Blepharospasms are primarily caused by abnormalities in the brain's basal ganglia, which controls movement, and can be triggered by fatigue and stress 1.

Possible Causes

  • Unknown causes: The exact cause of blepharospasms is often unknown
  • Brain abnormalities: Abnormalities in the brain's basal ganglia, which is the area responsible for controlling movement
  • Conditions: Blepharospasms can be a symptom of a condition such as blepharospasm disorder, dystonia, or Parkinson's disease
  • Medications: Certain medications like antipsychotics and anti-emetics can also cause blepharospasms as a side effect

Treatment

Treatment typically involves botulinum toxin injections, such as Botox, which can help relax the muscles and reduce spasms, as supported by the use of OnaBoNT-A and incoBoNT-A for treating blepharospasm 1. In some cases, medications like clonazepam or trihexyphenidyl may be prescribed to help manage symptoms. It's essential to consult a healthcare professional for proper diagnosis and treatment.

From the Research

Causes of Blepharospasm

  • The exact cause of blepharospasm is not fully understood, but it is believed to be related to abnormal functioning of the brain's motor control systems 2.
  • Research suggests that blepharospasm may be caused by a combination of factors, including abnormalities in the basal ganglia dopamine system, sensorimotor learning substrate, and patterns of increased blinking 2.
  • Some studies suggest that blepharospasm may be related to vascular compression of cranial nerve VII (CN VII), leading to involuntary unilateral contractions of muscles used in facial expression 3.
  • Other research suggests that blepharospasm may be caused by a dynamic circuit hypothesis, which incorporates the joint influence of motor cortical regions and direct descending projections from the substantia nigra pars reticulata on brainstem motor nuclei 2.

Pathophysiology of Blepharospasm

  • Blepharospasm is characterized by excessive or continuous eye closure related to overactivity of the orbicularis oculi and adjacent muscles bilaterally 4, 3.
  • The disorder can also involve involuntary movements of the lower face and neck muscles, as well as dystonic, uncontrolled movements in areas outside the facial nerve distribution 4.
  • Abnormal eye movements, including saccadic intrusions and extraocular muscle dystonia, can also occur in patients with blepharospasm 5.
  • The pathogenesis of blepharospasm is believed to involve abnormalities in the basal ganglia, brainstem structures, and sensorimotor learning substrate 2, 5.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

A Dynamic Circuit Hypothesis for the Pathogenesis of Blepharospasm.

Frontiers in computational neuroscience, 2017

Research

Botulinum toxin in the treatment of blepharospasm and hemifacial spasm.

Journal of neural transmission (Vienna, Austria : 1996), 2008

Research

Essential blepharospasm and related dystonias.

Survey of ophthalmology, 1989

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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