What is the mechanism behind relative bradycardia (slow heart rate) in typhoid fever?

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From the Guidelines

The mechanism behind relative bradycardia in typhoid fever is not explicitly explained by the provided evidence, but it is characterized by an insidious onset of sustained fever, headache, malaise, anorexia, relative bradycardia, and other symptoms 1. The relative bradycardia in typhoid fever is a complex phenomenon that involves the interplay of various factors, including the effects of Salmonella Typhi endotoxins on the cardiovascular system.

  • Typically, in febrile illnesses, the heart rate increases by about 10 beats per minute for each 1°C rise in temperature.
  • However, in typhoid fever, the heart rate is slower than expected for the degree of fever, which is known as relative bradycardia or pulse-temperature dissociation.
  • This occurs due to the stimulation of the vagus nerve by typhoid endotoxins, increasing parasympathetic tone to the heart, and potentially direct depression of the sinoatrial node function.
  • The release of cytokines like tumor necrosis factor-alpha and interleukin-6, triggered by the bacteria, may also affect cardiac function. The provided evidence 1 describes the clinical characteristics of typhoid fever, including relative bradycardia, but does not provide a detailed explanation of the underlying mechanism.
  • Relative bradycardia is considered a classic clinical sign of typhoid fever, although it can also occur in other infections.
  • It is most noticeable during the first week of illness and can be a valuable diagnostic clue when evaluating patients with prolonged fever of unknown origin.

From the Research

Mechanism of Relative Bradycardia in Typhoid Fever

The mechanism behind relative bradycardia in typhoid fever is not fully understood. However, several studies have proposed possible mechanisms, including:

  • Release of inflammatory cytokines 2
  • Increased vagal tone 2
  • Direct pathogenic effect on the myocardium 2
  • Electrolyte abnormalities 2

Pathogenesis of Relative Bradycardia

Relative bradycardia is found in a wide variety of infectious and noninfectious diseases, and its pathogenesis remains poorly understood 2. The incidence of this sign varies widely, which may be attributable to multiple factors, including population size, time course for measuring pulse and temperature, and lack of a consistent definition used 2.

Clinical Significance of Relative Bradycardia

Relative bradycardia is a sensitive but nonspecific clinical sign that may be an important bedside tool for narrowing the differential diagnosis of potential infectious and noninfectious etiologies 2. Recognizing this relationship may assist the clinician by providing bedside clinical clues into potential etiologies of disease, particularly in the setting of infectious diseases and in circumstances when other stigma of disease is absent 2.

Relative Bradycardia in Typhoid Fever

Relative bradycardia is a characteristic feature of typhoid fever, and it is often used as a clinical sign for diagnosis 3, 4. The classic findings of typhoid fever include rose spots, relative bradycardia, and stepwise fevers, but unfortunately, these signs are frequently absent 4.

Treatment of Typhoid Fever

The treatment of typhoid fever typically involves the use of antibiotics, such as fluoroquinolones, ceftriaxone, and azithromycin 4, 5. The choice of antibiotic depends on the severity of the disease, the presence of multidrug resistance, and the patient's clinical condition 5.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

The Clinical Significance of Relative Bradycardia.

WMJ : official publication of the State Medical Society of Wisconsin, 2018

Research

Relative bradycardia in infectious diseases.

The Journal of infection, 1996

Research

Current trends in typhoid Fever.

Current gastroenterology reports, 2003

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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