Can Repatha (evolocumab) cause hypertriglyceridemia?

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Last updated: May 28, 2025View editorial policy

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From the Guidelines

Repatha (evolocumab) does not typically cause hypertriglyceridemia; in fact, it may slightly reduce triglyceride levels in some patients. Repatha is a PCSK9 inhibitor primarily designed to lower LDL cholesterol by increasing the liver's ability to remove LDL from the bloodstream. While its main effect is on LDL cholesterol, clinical studies, such as the FOURIER trial 1, have shown that Repatha can modestly decrease triglyceride levels. The FOURIER trial demonstrated a 16.2% decrease in triglyceride levels with evolocumab compared to a 0.3% increase with placebo. If a patient taking Repatha experiences elevated triglycerides, other factors are likely responsible, such as diet (high carbohydrate or alcohol intake), obesity, uncontrolled diabetes, certain medications (like estrogens, corticosteroids, or beta-blockers), or genetic predisposition. Any significant increase in triglycerides while on Repatha should prompt evaluation of these alternative causes rather than attributing the effect to the medication itself. Patients concerned about triglyceride levels should discuss their complete lipid profile with their healthcare provider.

Some key points to consider when evaluating triglyceride levels in patients taking Repatha include:

  • Dietary counseling and weight loss in patients who are overweight or obese, as recommended by the Endocrine Society 1
  • Reduced intake of dietary fat and simple carbohydrates in combination with drug treatment to reduce the risk of pancreatitis in patients with severe to very severe hypertriglyceridemia
  • Consideration of fibrates, niacin, and n-3 fatty acids, alone or in combination, in patients with moderate to severe hypertriglyceridemia
  • Statins may have a modest triglyceride-lowering effect, but should not be used alone in patients with severe or very severe hypertriglyceridemia.

Overall, Repatha is not typically associated with hypertriglyceridemia, and any increases in triglyceride levels should be evaluated in the context of other potential causes.

From the Research

Repatha and Hypertriglyceridemia

  • There is no direct evidence in the provided studies that Repatha (evolocumab) causes hypertriglyceridemia 2, 3, 4, 5, 6.
  • The studies focus on the efficacy and safety of evolocumab in reducing low-density lipoprotein cholesterol (LDL-C) levels and its effects on cardiovascular outcomes.
  • Some studies mention the effects of evolocumab on other lipid parameters, including triglycerides, but do not suggest that it causes hypertriglyceridemia 4, 6.
  • In fact, one study notes that treatment with evolocumab significantly improves levels of triglycerides, among other lipid parameters 6.

Mechanism of Action

  • Evolocumab is a monoclonal antibody that inhibits proprotein convertase subtilisin/kexin type 9 (PCSK9), leading to a reduction in LDL-C levels 3, 4, 6.
  • The mechanism of action of evolocumab does not suggest that it would cause an increase in triglyceride levels.

Safety and Efficacy

  • The studies provide evidence for the safety and efficacy of evolocumab in reducing LDL-C levels and improving cardiovascular outcomes 2, 3, 4, 5, 6.
  • There is no mention of hypertriglyceridemia as a side effect or adverse event associated with evolocumab treatment in the provided studies.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Evolocumab: A Review in Hyperlipidemia.

American journal of cardiovascular drugs : drugs, devices, and other interventions, 2016

Research

Long-Term Efficacy and Safety of Evolocumab in Patients With Hypercholesterolemia.

Journal of the American College of Cardiology, 2019

Research

Evolocumab: Considerations for the Management of Hyperlipidemia.

Current atherosclerosis reports, 2018

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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