What is the etiology of paroxysmal supraventricular tachycardia (SVT)?

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From the Guidelines

Paroxysmal supraventricular tachycardia (PSVT) is primarily caused by reentry circuits in the heart, with atrioventricular nodal reentrant tachycardia (AVNRT) being the most common mechanism, accounting for about 60% of cases, as stated in the most recent guidelines 1.

Etiology of PSVT

The etiology of PSVT can be attributed to various mechanisms, including:

  • Reentry circuits, which are the most common cause
  • Abnormal automaticity, where tissues exhibit enhanced diastolic phase 4 depolarization, leading to an increase in firing rate compared to pacemaker cells 1
  • Triggered activity, associated with disturbances of recovery or repolarization, generating afterdepolarizations that can trigger early action potentials 1

Common Causes of PSVT

The most common causes of PSVT include:

  • Atrioventricular nodal reentrant tachycardia (AVNRT), accounting for about 60% of cases
  • Accessory pathway-mediated tachycardias, particularly Wolff-Parkinson-White syndrome, representing about 30% of cases
  • Atrial tachycardia, causing approximately 10% of PSVT cases, stemming from an ectopic focus in the atria that fires rapidly

Risk Factors for Developing PSVT

Risk factors for developing PSVT include:

  • Congenital heart defects
  • Coronary artery disease
  • High blood pressure
  • Thyroid disorders
  • Caffeine consumption
  • Alcohol use
  • Certain medications

Presentation and Treatment of PSVT

PSVT typically presents with sudden onset of rapid heartbeat, palpitations, chest discomfort, shortness of breath, dizziness, and occasionally syncope. While most episodes are benign and self-limiting, recurrent episodes may require treatment with medications like beta-blockers, calcium channel blockers, or antiarrhythmics, or definitive treatment with catheter ablation for persistent cases, as recommended in the guidelines 1.

From the Research

Etiology of Paroxysmal SVT

  • Paroxysmal supraventricular tachycardia (PSVT) is caused by reentry, and the tachycardias are classified electrophysiologically according to the anatomic location of the reentry circuit 2
  • The most common form of PSVT is atrioventricular nodal reentry, which occurs when there are two conducting pathways with different conduction times and refractory periods in the atrioventricular node 2
  • Another common form of PSVT is atrioventricular reciprocating tachycardia, which depends on an anatomically distinct accessory pathway that conducts impulses between the atria and ventricles, bypassing the AV node 2
  • PSVT can also be caused by focal atrial tachycardia, which is a less common type of supraventricular tachycardia 3

Mechanisms of PSVT

  • Reentry is the primary mechanism of PSVT, which occurs when an electrical impulse circulates repeatedly in a closed circuit, causing a rapid heart rate 2, 4
  • The presence of inhomogeneous, accessory, or concealed conducting pathways can contribute to the development of reentry and PSVT 4
  • The anatomic location of the reentry circuit can affect the type of PSVT and its treatment 2, 3

Types of PSVT

  • Atrioventricular nodal reentrant tachycardia (AVNRT) is the most common type of PSVT, accounting for approximately 50-60% of cases 5, 3
  • Atrioventricular reentrant tachycardia (AVRT) is another common type of PSVT, which uses an accessory pathway to conduct impulses between the atria and ventricles 2, 5
  • Focal atrial tachycardia is a less common type of PSVT, which originates from a single focus in the atria 3

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Paroxysmal supraventricular tachycardias.

The Journal of emergency medicine, 1996

Research

[Paroxysmal supraventricular tachycardia-mechanisms, diagnostic, and treatment].

Therapeutische Umschau. Revue therapeutique, 2014

Research

Paroxysmal Supraventricular Tachycardia: Pathophysiology, Diagnosis, and Management.

Critical care nursing clinics of North America, 2016

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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