What is Kallmann syndrome?

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Last updated: May 29, 2025View editorial policy

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From the Research

Kallmann syndrome is a genetic disorder characterized by delayed or absent puberty and a reduced or absent sense of smell (anosmia), and the most effective management strategy involves hormone replacement therapy to induce puberty and maintain secondary sexual characteristics, as well as genetic molecular diagnostics to reduce the risk of the syndrome in future generations 1.

Key Features of Kallmann Syndrome

  • Hypogonadotropic hypogonadism due to lack of production of sex hormones
  • Anosmia or hyposmia (reduced or absent sense of smell)
  • Delayed or absent puberty
  • Associated features may include midline facial defects, skeletal abnormalities, kidney problems, and neurological symptoms

Diagnosis and Management

  • Genetic molecular diagnostics through prenatal diagnosis and preimplantation genetic testing are significant in reducing the risk of Kallmann syndrome in the next generation 1
  • Hormone replacement therapy is the primary treatment approach, with testosterone replacement for males and estrogen therapy for females 1, 2
  • Fertility treatments with gonadotropins or pulsatile GnRH therapy may be beneficial for patients wishing to conceive 3

Importance of Early Diagnosis and Treatment

  • Early diagnosis and treatment are crucial to prevent complications like osteoporosis and to support normal physical and psychological development 1, 4
  • Treatment should aim to induce and maintain secondary sex characteristics, build and sustain bone density, and restore fertility when indicated 1, 2

References

Research

Kallmann syndrome: Diagnostics and management.

Clinica chimica acta; international journal of clinical chemistry, 2025

Research

Kallmann syndrome in women: from genes to diagnosis and treatment.

Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology, 2013

Research

Clinical evaluation in isolated hypogonadotrophic hypogonadism (Kallmann syndrome).

Journal of pediatric endocrinology & metabolism : JPEM, 1998

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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