What are the types of intracranial hemorrhages and their associated blood vessels?

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From the Guidelines

Intracranial hemorrhages are primarily classified into four categories: noncoagulopathic spontaneous intracranial hemorrhage (ICH), coagulopathic spontaneous ICH, noncoagulopathic traumatic intracranial hemorrhage, and coagulopathic traumatic intracranial hemorrhage, with each type having distinct vascular pathologies and locations of bleeding. These categories are based on the underlying cause of the hemorrhage, with spontaneous ICH often resulting from hypertension-related rupture of small penetrating arteries, such as the lenticulostriate arteries supplying the basal ganglia 1. Traumatic intracranial hemorrhages, on the other hand, can result from various mechanisms, including tears in the middle meningeal artery, rupture of bridging veins, or other injuries to the blood vessels in the brain 1. The management of each type of intracranial hemorrhage requires a specific approach, taking into account the underlying vascular pathology and location of bleeding, as well as the presence of any coagulopathy or other complicating factors 1. Some of the key blood vessels involved in intracranial hemorrhages include:

  • Middle meningeal artery, which is commonly affected in epidural hemorrhages
  • Bridging veins, which are often involved in subdural hemorrhages
  • Berry aneurysms in the Circle of Willis, particularly at the anterior communicating artery and posterior communicating artery junctions, which are a common cause of subarachnoid hemorrhages
  • Lenticulostriate arteries, which are small penetrating arteries that supply the basal ganglia and are often affected in hypertensive intraparenchymal hemorrhages
  • Choroid plexus, which can be a source of bleeding in intraventricular hemorrhages. The most critical factor in managing intracranial hemorrhages is to promptly identify the type and location of the bleed, as well as any underlying coagulopathy or other complicating factors, in order to provide targeted and effective treatment.

From the Research

Types of Intracranial Hemorrhages

  • Epidural hemorrhage: typically caused by trauma, associated with middle meningeal artery injury 2
  • Subdural hemorrhage: increasingly common in older adults, often caused by trauma or spontaneous bleeding 3
  • Subarachnoid hemorrhage: can be caused by traumatic lesions of meningeal arteries or spontaneous rupture of cerebral aneurysms 2
  • Intraparenchymal hemorrhage: often associated with hypertension, especially in deep locations such as the basal ganglia, pons, or caudate nucleus 4, 5

Associated Vessels

  • Middle meningeal artery: associated with epidural hemorrhage and traumatic aneurysms 2
  • Deep perforator arteries: associated with hypertensive arteriopathy and intraparenchymal hemorrhage 5
  • Cerebral arteries: associated with subarachnoid hemorrhage and cerebral aneurysms 2

Causes and Risk Factors

  • Trauma: a common cause of intracranial hemorrhage, especially in younger adults 4, 2
  • Hypertension: a major risk factor for intraparenchymal hemorrhage, especially in deep locations 4, 5
  • Vascular malformations: can cause intracranial hemorrhage, especially in younger adults 6
  • Anticoagulant therapy: can increase the risk of intracranial hemorrhage, especially in older adults 3, 6

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Updates on the diagnosis and management of subdural hematoma.

JAAPA : official journal of the American Academy of Physician Assistants, 2024

Research

Intracerebral hemorrhage: an update on diagnosis and treatment.

Expert review of neurotherapeutics, 2019

Research

Intracranial hemorrhage.

American journal of respiratory and critical care medicine, 2011

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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