What is the treatment for Hepatitis C (HCV)?

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Last updated: June 5, 2025View editorial policy

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From the Guidelines

The treatment for Hepatitis C (HCV) involves direct-acting antiviral (DAA) medications taken for 8-12 weeks, with cure rates exceeding 95%, and the standard first-line regimens include combinations like sofosbuvir/velpatasvir (Epclusa) or glecaprevir/pibrentasvir (Mavyret) 1.

Key Considerations

  • The choice of treatment regimen depends on factors such as viral genotype, liver function, prior treatment history, and comorbidities.
  • Before starting treatment, patients need assessment of liver fibrosis and screening for potential drug interactions.
  • Treatment is typically one pill daily with minimal side effects like headache, fatigue, and nausea.
  • Regular monitoring during treatment includes blood tests to assess viral load and liver function.

Treatment Goals

  • The goal is to achieve sustained virologic response (SVR), defined as undetectable HCV RNA 12 weeks after completing therapy, which indicates cure.
  • These medications work by targeting specific proteins essential for viral replication, preventing the virus from multiplying and allowing the body to clear the infection.

Special Considerations

  • For patients with decompensated cirrhosis or other complications, treatment should be managed by specialists and may require adjusted regimens 1.
  • Patients with recently acquired de novo hepatitis C should be treated with the combination of sofosbuvir and velpatasvir or with the combination of glecaprevir and pibrentasvir for 8 weeks 1.

Patient Education

  • Patients with HCV should receive counseling about transmission prevention, such as avoidance of sharing personal hygiene articles and avoidance of needle sharing in the setting of intravenous drug use 1.
  • Patients do not need to avoid acetaminophen, although it is advisable to set a lower maximum daily dosage of 2 g rather than 4 g in patients with cirrhosis related to HCV 1.

From the FDA Drug Label

The overall SVR12 rate was 98% and no subjects experienced virologic failure. The presence of renal impairment did not affect efficacy; no dose-adjustments were required during the trial. In EXPEDITION-2, the SVR12 rate in HCV/HIV-1 co-infected subjects was 98% (150/153). The overall SVR12 rate in post-transplant subjects was 98% (98/100). The overall SVR12 rate was 98% in former/non-PWID subjects and 89% in current/recent PWID subjects;

The treatment for Hepatitis C (HCV) is MAVYRET (glecaprevir and pibrentasvir).

  • Treatment duration: 8,12, or 16 weeks, depending on the patient's condition and history of treatment.
  • Patient populations:
    • Treatment-naïve or PRS treatment-experienced adults without cirrhosis or with compensated cirrhosis.
    • Adults with HCV/HIV-1 coinfection without cirrhosis or with compensated cirrhosis.
    • Adults with liver or kidney transplant without cirrhosis.
    • People who inject drugs (PWID) and those on medication-assisted treatment (MAT) for opioid use disorder.
  • Efficacy: High SVR12 rates (89-98%) were observed in different patient populations. 2

From the Research

Treatment for Hepatitis C

The treatment for Hepatitis C (HCV) has undergone significant advancements in recent years.

  • Direct-acting antivirals (DAAs) have revolutionized the treatment of HCV, offering high sustained virological response (SVR) rates and shorter treatment durations 3.
  • Several oral regimens combining DAAs from different families have been developed, resulting in SVR rates above 90% and treatment durations of 12 weeks or less 3.
  • Real-world effectiveness studies have demonstrated high SVR rates for various DAA regimens, including sofosbuvir/velpatasvir and daclatasvir plus sofosbuvir, in patients with genotype 2 and 3 infections 4, 5.

Direct-Acting Antiviral Regimens

  • Sofosbuvir/velpatasvir is a pangenotypic DAA combination that has been shown to be effective in treating patients with HCV genotype 1 through 6 6.
  • Daclatasvir plus sofosbuvir and velpatasvir/sofosbuvir have been found to produce similar SVR rates in patients with genotype 2 and 3 infections 5.
  • Sofosbuvir/velpatasvir/voxilaprevir is a DAA regimen that has been shown to be effective in treating DAA-experienced patients with genotype 1-4 infections 7.

Factors Affecting Treatment Outcomes

  • Prior treatment experience and advanced liver disease have been found to be significant predictors of reduced SVR rates in patients with genotype 3 infections 5.
  • The presence of cirrhosis and prior NS5A experience have also been found to affect treatment outcomes in DAA-experienced patients 7.
  • The choice of DAA regimen does not appear to have a significant impact on SVR rates in patients with genotype 2 and 3 infections 5.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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