Primary Aldosteronism: This condition is characterized by the excessive production of aldosterone, leading to hypertension, hypokalemia (low potassium levels), and metabolic alkalosis (high HCO3 levels). The sodium level is often at the higher end of the normal range or slightly elevated, which aligns with the patient's sodium level of 137.

Renal Artery Stenosis: This condition can lead to secondary aldosteronism due to activation of the renin-angiotensin-aldosterone system, resulting in similar lab findings.
Cushing's Syndrome: Excess cortisol can cause hypertension and hypokalemia, and while it more commonly causes metabolic alkalosis, the presentation can vary.
Liddle's Syndrome: A rare genetic disorder but considered here due to its similarity in presentation with primary aldosteronism, characterized by excessive sodium absorption and potassium wasting in the kidneys.

Pheochromocytoma: Although less likely to present with hypokalemia and high HCO3 as primary features, it's a critical diagnosis to consider due to its potential for severe, life-threatening hypertension.
Licorice-Induced Pseudohyperaldosteronism: Certain compounds in licorice can mimic the effects of aldosterone, leading to hypertension, hypokalemia, and metabolic alkalosis.

Bartter Syndrome: Typically presents in childhood with hypokalemia, metabolic alkalosis, and normal to low blood pressure, but some variants could potentially present later in life with hypertension due to secondary effects.
Gitelman Syndrome: Similar to Bartter Syndrome, it usually presents with hypokalemia and metabolic alkalosis but typically with normal or low blood pressure. However, it's a consideration in the differential due to its effects on potassium and acid-base balance.