What are the rare causes of leptomeningeal (inflammation of the leptomeninges) enhancement?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 25, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the Guidelines

Leptomeningeal enhancement has several rare causes beyond the common etiologies of infection, malignancy, and inflammatory conditions, including neurosarcoidosis, intravascular lymphoma, Vogt-Koyanagi-Harada syndrome, hypertrophic pachymeningitis, superficial siderosis, neurocutaneous melanosis, and IgG4-related disease, as reported in various studies 1. These uncommon causes should be considered in cases where leptomeningeal enhancement is present but common causes have been excluded. Some key points to consider include:

  • Neurosarcoidosis can present with leptomeningeal involvement in 5-10% of cases 1
  • Intravascular lymphoma is a rare subtype of non-Hodgkin lymphoma that can affect the leptomeninges 1
  • Vogt-Koyanagi-Harada syndrome is an autoimmune disorder affecting melanocyte-rich tissues that can cause leptomeningeal enhancement particularly at the posterior fossa 1
  • Hypertrophic pachymeningitis is a condition characterized by thickening of the dura mater that may occasionally extend to involve the leptomeninges 1
  • Superficial siderosis can mimic leptomeningeal enhancement on MRI due to hemosiderin deposition 1
  • Neurocutaneous melanosis is a rare congenital disorder that may present with leptomeningeal melanosis that enhances on contrast imaging 1
  • IgG4-related disease is a systemic fibroinflammatory condition that can rarely affect the leptomeninges 1 Diagnosis typically requires correlation with clinical presentation, laboratory findings, and sometimes biopsy for definitive identification, as emphasized in recent guidelines 1. It is essential to consider these rare causes and to use a combination of clinical, laboratory, and imaging findings to establish a diagnosis, as highlighted in the most recent study 1.

From the Research

Rare Causes of Leptomeningeal Enhancement

The following are rare causes of leptomeningeal enhancement:

  • Primary leptomeningeal lymphoma, including ALK-positive anaplastic large cell lymphoma (ALCL) and primary effusion lymphoma (PEL) 2
  • MOG-IgG-associated encephalomyelitis, which can present with bilateral symmetrical deep gray matter involvement and leptomeningeal enhancement 3
  • Neurosarcoidosis, which can mimic malignancy and present with leptomeningeal enhancement 4
  • Leptomeningeal myelomatosis, a rare complication of multiple myeloma characterized by widespread nodular leptomeningeal enhancement 5
  • Leptomeningeal melanocytosis, a rare cause of seizure in the pediatric population characterized by progression of leptomeningeal enhancement 6

Characteristics of Rare Causes

Some key characteristics of these rare causes include:

  • Primary leptomeningeal lymphoma: exclusive leptomeningeal disease, lymphocytic pleocytosis, and brisk mitotic activity 2
  • MOG-IgG-associated encephalomyelitis: bilateral symmetrical deep gray matter involvement, leptomeningeal enhancement, and positive serum MOG IgG 3
  • Neurosarcoidosis: lymphocytic pleocytosis, hypoglycorrhachia, and hyperproteinorrachia, with noncaseating granulomatous inflammation on biopsy 4
  • Leptomeningeal myelomatosis: widespread nodular leptomeningeal enhancement, involvement of supra- and sub-tentorial regions, and poor prognosis 5
  • Leptomeningeal melanocytosis: progression of leptomeningeal enhancement, increased seizure activity, and confirmation of diagnosis by biopsy 6

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.