What is the general pathophysiology of cancer?

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From the Guidelines

Cancer develops through a multistep process involving genetic and epigenetic alterations that disrupt normal cellular function, leading to uncontrolled growth and the potential to invade surrounding tissues and spread to distant sites through metastasis. The pathophysiology begins with mutations in critical genes that control cell growth, division, and death, as noted in studies such as 1, which highlights the role of genetic mutations in cancer development. These mutations may occur in proto-oncogenes, which become hyperactivated oncogenes that drive excessive cell proliferation, or in tumor suppressor genes, which normally restrict cell growth but lose their protective function when mutated. DNA repair genes may also be damaged, leading to genomic instability and accelerated mutation accumulation, a concept supported by research like 1, which discusses the processes of tumor initiation and progression in lung cancer.

Key aspects of cancer pathophysiology include:

  • Genetic alterations: Mutations in genes controlling cell growth, division, and death, as discussed in 1 and 1.
  • Epigenetic alterations: Changes in gene expression without altering the DNA sequence, such as methylation or acetylation, mentioned in 1.
  • Hallmark capabilities of cancer cells: Sustaining proliferative signaling, evading growth suppressors, resisting cell death, enabling replicative immortality, inducing angiogenesis, and activating invasion and metastasis pathways, as outlined in the example answers.
  • Tumor microenvironment: Cancer cells often reprogram their energy metabolism and recruit normal cells to create a supportive environment, as noted in the example answers.

The specific mutations and altered pathways vary widely among different cancer types, explaining the diverse behaviors and treatment responses observed clinically, a point emphasized across various studies including 1 and 1. Understanding these variations is crucial for developing effective treatment strategies tailored to the specific characteristics of each cancer type.

From the Research

General Pathophysiology of Cancer

The general pathophysiology of cancer involves a complex series of genetic and epigenetic changes that ultimately lead to uncontrolled cell growth and tumor formation.

  • Cancer development is a multi-step process that requires the sequential acquisition of mutations in genes responsible for maintaining genomic integrity, as well as the activation of oncogenes and the inactivation of tumor suppressor genes 2.
  • The process of carcinogenesis can be divided into at least three stages: initiation, promotion, and progression, with each stage involving distinct molecular and cellular changes 3, 4, 5.
  • Initiation involves an irreversible genetic alteration, such as a mutation or deletion in DNA, while promotion involves reversible changes in gene expression mediated through promoter-receptor interactions 5.
  • Progression is characterized by karyotypic instability and malignant growth, and is often associated with the inactivation of tumor suppressor genes and the activation of oncogenes 3, 5.

Key Molecular Targets

Critical molecular targets during the stages of carcinogenesis include:

  • Proto-oncogenes and cellular oncogenes, which can be activated by mutations or other genetic alterations 5.
  • Tumor suppressor genes, which can be inactivated by mutations or epigenetic changes, such as DNA methylation or histone modification 3, 6.
  • MicroRNAs, which can regulate gene expression and contribute to cancer development and progression 6.

Epigenetic Changes

Epigenetic changes, such as DNA methylation, histone modification, and non-coding RNA expression, can also play a crucial role in cancer development and progression 6.

  • These changes can modify gene expression and contribute to the activation of oncogenes and the inactivation of tumor suppressor genes.
  • Epigenetic changes can also influence the epithelial-mesenchymal transition, a process that can contribute to cancer metastasis 6.

Metastasis

Metastasis requires a series of complex cellular and molecular changes, including:

  • Down-regulation of cell adhesion receptors necessary for tissue-specific, cell-cell attachment 6.
  • Up-regulation of receptors that enhance cell motility and invasion 6.
  • Angiogenesis, matrix degradation, and evasion of the host immune response are also critical steps in the metastatic process 4.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

The molecular biology of cancer.

Molecular aspects of medicine, 2000

Research

Carcinogenesis: basic principles.

Drug and chemical toxicology, 1996

Research

[Carcinogenesis].

Salud publica de Mexico, 2011

Research

Cancer development, progression, and therapy: an epigenetic overview.

International journal of molecular sciences, 2013

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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