Can Chronic Kidney Disease (CKD) cause hyponatremia?

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Last updated: February 27, 2025View editorial policy

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From the Guidelines

Chronic Kidney Disease (CKD) can indeed cause hyponatremia, a condition characterized by low sodium levels in the blood, as evidenced by the high incidence of electrolyte disorders, including hyponatremia, in patients with CKD 1. The pathophysiology behind CKD-induced hyponatremia involves the kidneys' impaired ability to regulate fluid and electrolyte balance, leading to dilutional hyponatremia due to excess water retention. Key factors contributing to hyponatremia in CKD patients include:

  • Impaired urinary diluting capacity, making it difficult for the kidneys to excrete free water
  • Medications such as diuretics (especially thiazides) that increase sodium excretion
  • Decreased dietary intake and malnutrition, which can further affect sodium levels Management of hyponatremia in CKD patients typically involves:
  • Addressing the underlying CKD
  • Fluid restriction (often 1-1.5 liters daily)
  • Possible sodium supplementation in severe cases
  • Careful medication review to minimize the use of sodium-excreting medications Regular monitoring of sodium levels is crucial in CKD patients, especially those with advanced disease or on medications that affect sodium balance, as highlighted in the ESPEN guideline on clinical nutrition in hospitalized patients with acute or chronic kidney disease 1.

From the Research

Relationship Between Chronic Kidney Disease (CKD) and Hyponatremia

  • CKD can lead to hyponatremia due to the kidneys' reduced ability to regulate electrolyte and water balance 2.
  • In advanced CKD, the range of urine osmolality approaches plasma osmolality, leading to water overload and potentially causing hyponatremia 2.
  • Hyponatremia is associated with fluid imbalance and adverse renal outcomes in CKD patients treated with diuretics 3.
  • The presence of hyponatremia in CKD patients is a prognostic indicator for adverse outcomes, including increased risk of renal replacement therapy 3.

Mechanisms and Clinical Implications

  • The failing kidney's gradual development of hyposthenuria and isosthenuria predisposes CKD patients to hypo- and hypernatremia 4.
  • Dysnatremias, including hyponatremia, are common in CKD patients and impart an increased risk of mortality 5.
  • The management of patients with CKD and marked abnormalities in serum sodium concentrations requires specific recommendations for modifications in renal replacement therapy prescription 5.

Association with Mortality

  • Hyponatremia is a prognostic marker for mortality, and its association with mortality is consistent across progressive stages of CKD and acute kidney injury (AKI) 6.
  • The contribution of kidney failure to the pathophysiology of hyponatremia does not alter its association with mortality 6.
  • CKD and AKI patients with hyponatremia have a higher prevalence of mortality, and hyponatremia is associated with increased hazard ratios for death in these cohorts 6.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

[Electrolyte and acid-base balance disorders in advanced chronic kidney disease].

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia, 2008

Research

Significance of hypo- and hypernatremia in chronic kidney disease.

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 2012

Research

Dysnatremias in patients with kidney disease.

American journal of kidney diseases : the official journal of the National Kidney Foundation, 2014

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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