What are the causes of acute mitral regurgitation (mitral valve insufficiency)?

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Last updated: February 27, 2025View editorial policy

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From the Guidelines

Acute mitral regurgitation is primarily caused by conditions that suddenly disrupt the normal function of the mitral valve apparatus, with the most common cause being acute myocardial infarction, particularly involving the inferior or posterior wall of the left ventricle, which can lead to papillary muscle dysfunction or rupture, as noted in 1 and further supported by 1. The causes of acute mitral regurgitation can be categorized into several key areas, including:

  • Acute myocardial infarction, which can lead to mitral valve annulus dilatation due to left ventricular dilatation and dysfunction, papillary muscle dysfunction, or papillary muscle rupture, as described in 1
  • Infective endocarditis, where bacterial infection damages the valve leaflets or chordae tendineae, as mentioned in 1
  • Trauma to the chest, resulting in damage to the valve apparatus or papillary muscles
  • Spontaneous chordal rupture, often associated with myxomatous degeneration or mitral valve prolapse, as noted in 1
  • Prosthetic valve dysfunction, including paravalvular leaks or structural valve deterioration, as mentioned in 1 It is essential to promptly identify the cause of acute mitral regurgitation, as it can lead to rapid hemodynamic deterioration, pulmonary edema, and cardiogenic shock due to the sudden volume overload on a left ventricle that hasn't had time to compensate, unlike in chronic mitral regurgitation where adaptive mechanisms develop gradually, as supported by the findings in 1 and 1.

From the Research

Causes of Acute Mitral Regurgitation

The causes of acute mitral regurgitation (mitral valve insufficiency) can be classified into several categories, including:

  • Chordae tendineae (CT) rupture, which is the most common etiology of acute severe mitral regurgitation today, especially in patients with floppy mitral valve/mitral valve prolapse 2
  • Papillary muscle (PM) rupture, often associated with myocardial infarction (AMI) 2, 3, 4, 5
  • Infective endocarditis (IE), which can cause native or prosthetic valve damage, including leaflet perforation, ring detachment, and CT or PM rupture 2, 6
  • Mitral valve prosthesis dysfunction, which can be biological or mechanical 6
  • Trauma, such as percutaneous valvuloplasty 6

Mechanisms and Clinical Presentation

The mechanisms of acute mitral regurgitation can be classified into four categories:

  • Mutilating valve lesions
  • Rupture of chordae tendineae
  • Papillary muscle lesions
  • Biological or mechanical valve prosthesis dysfunction The clinical presentation and prognosis of acute mitral regurgitation depend on the volume of regurgitation, left ventricular function, and left atrial compliance 6

Diagnosis and Treatment

Diagnosis of acute mitral regurgitation can be confirmed by Doppler ultrasound, which defines the mechanism and sometimes eliminates the need for an invasive investigation 2, 6 Treatment of acute mitral regurgitation depends on the aetiology, volume of regurgitation, left ventricular function, and treatment implemented, and may include:

  • Vasodilators, such as sodium nitroprussate infusion 6
  • Intra-aortic counterpulsation 6
  • Emergency surgery, which may consist of valve replacement or surgical repair 2, 6, 5

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Mitral regurgitation after myocardial infarction. Coronary artery bypass grafting and mitral valve replacement with chordae preservation.

The Japanese journal of thoracic and cardiovascular surgery : official publication of the Japanese Association for Thoracic Surgery = Nihon Kyobu Geka Gakkai zasshi, 2001

Research

[Acute mitral valve insufficiency].

Annales de cardiologie et d'angeiologie, 1995

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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