What is the Epstein-Barr Virus (EBV) Antibody (Ab)/Viral Capsid Antigen (VCA) Immunoglobulin G (IgG) test?

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EBV Ab/VCA IgG Test

The EBV Ab/VCA IgG test is a serological test that detects immunoglobulin G antibodies against the viral capsid antigen of Epstein-Barr virus, primarily used to determine past exposure to EBV infection. 1

What This Test Measures

The EBV Ab/VCA IgG test specifically measures antibodies that target the viral capsid antigen (VCA) of Epstein-Barr virus. This test is part of a comprehensive EBV serological panel that helps differentiate between:

  • Recent primary infection
  • Past infection
  • Reactivation of latent infection

Clinical Significance

Interpretation of Results

  • Positive VCA IgG: Indicates current or past EBV infection

    • Over 90% of the normal adult population has IgG class antibodies to VCA antigens 1
    • VCA IgG antibodies develop during acute infection and persist for life
    • The presence of VCA IgG alone (without VCA IgM and with EBNA antibodies) typically indicates past infection
  • Negative VCA IgG: Suggests no prior exposure to EBV

Diagnostic Algorithm for EBV Infection Status

To properly interpret EBV Ab/VCA IgG results, they should be evaluated alongside other EBV markers:

  1. Recent primary infection pattern:

    • Positive VCA IgM
    • Positive VCA IgG (often with low avidity)
    • Negative EBNA antibodies 1
  2. Past infection pattern:

    • Negative VCA IgM
    • Positive VCA IgG (high avidity)
    • Positive EBNA antibodies 1
  3. Late primary infection or reactivation pattern:

    • Positive VCA IgM
    • Positive VCA IgG
    • Positive EBNA antibodies
    • In this case, IgG avidity testing is crucial to differentiate between late primary infection (low avidity) and reactivation (high avidity) 2

Clinical Applications

The EBV Ab/VCA IgG test is particularly useful in:

  • Diagnosing infectious mononucleosis when heterophile antibody tests (Monospot) are negative (occurs in approximately 10% of patients, especially children under 10 years) 1
  • Differentiating EBV infection from other causes of mononucleosis-like illness (CMV, adenovirus, HIV, Toxoplasma gondii)
  • Determining immune status to EBV
  • Evaluating patients with suspected EBV-associated lymphoproliferative disorders

Important Considerations and Pitfalls

  • Timing matters: VCA IgG antibodies typically develop within 1-2 weeks after symptom onset and persist for life 1
  • Avidity testing: When both VCA IgG and IgM are present, avidity testing of VCA IgG can help distinguish between recent primary infection (low avidity) and past infection/reactivation (high avidity) 2
  • False positives: Cross-reactivity with other herpesvirus infections can occur
  • Interpretation challenges: The combination of positive VCA IgG, positive VCA IgM, and positive EBNA-1 antibodies can occur in both late primary infection and during subclinical viral reactivation 3
  • Test limitations: Approximately 5-10% of patients who have been infected with EBV fail to develop antibodies to the EBNA antigen, which can complicate interpretation 1

Specimen Collection and Handling

  • Specimen type: Serum (clot tube)
  • Transport conditions: Room temperature
  • Optimal transport time: Less than 2 hours 1

The EBV Ab/VCA IgG test is an essential component of EBV serological testing and, when interpreted correctly alongside other EBV markers, provides valuable diagnostic information about a patient's EBV infection status.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Prevalence of primary versus reactivated Epstein-Barr virus infection in patients with VCA IgG-, VCA IgM- and EBNA-1-antibodies and suspected infectious mononucleosis.

Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology, 2007

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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