How are Glucagon-like peptide-1 (GLP-1) receptor agonists made?

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Last updated: March 3, 2025View editorial policy

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From the Guidelines

GLP-1 receptor agonists are manufactured through recombinant DNA technology, where bacteria or yeast cells are genetically engineered to produce proteins that mimic human glucagon-like peptide-1 (GLP-1). The process involves inserting the gene for the desired GLP-1 analog into host cells, which then produce the protein in large quantities 1. These proteins are then harvested, purified, and formulated into injectable medications. Some common GLP-1 agonists include semaglutide, liraglutide, dulaglutide, and exenatide, which are delivered by subcutaneous injection and stimulate insulin secretion and reduce glucagon secretion in a glucose-dependent manner 1. The manufacturing process often involves modifications to the natural GLP-1 molecule to extend its half-life in the body, as natural GLP-1 is rapidly degraded by enzymes. For example, structural differences among GLP-1 receptor agonists, such as dulaglutide, exenatide extended-release, and semaglutide, affect duration of action, and their formulation and dosing may affect efficacy for glucose-lowering and weight reduction as well as side effect profile and cardiovascular effects 1. Key characteristics of GLP-1 receptor agonists include:

  • High glucose-lowering efficacy with minimal risk for hypoglycemia
  • Variation in duration of action and formulation, affecting efficacy and side effects
  • Administration via subcutaneous injection, with varying frequencies (e.g., once weekly for semaglutide, dulaglutide, and exenatide extended-release, and once daily for liraglutide and lixisenatide) 1.

From the FDA Drug Label

The peptide backbone is produced by yeast fermentation. The peptide precursor of liraglutide, produced by a process that includes expression of recombinant DNA in Saccharomyces cerevisiae,

GLP-1 receptor agonists are made through a process involving:

  • Yeast fermentation to produce the peptide backbone, as seen in semaglutide 2
  • Recombinant DNA expression in Saccharomyces cerevisiae to produce the peptide precursor, as seen in liraglutide 3 These processes are used to create the foundation for the GLP-1 receptor agonist molecules.

From the Research

Manufacturing of GLP-1 Receptor Agonists

  • GLP-1 receptor agonists are produced using various methods, including recombinant DNA technology 4, 5.
  • The manufacturing process for semaglutide, a GLP-1 receptor agonist, involves multistep synthesis and can be challenging due to its large peptide size and hydrophobic fatty acid side chain 6.
  • Researchers have developed alternative analogues, such as a glycosylated GLP-1 analogue, which can be produced in higher yields and has similar biological activity to semaglutide 6.
  • GLP-1 receptor agonists can be formulated for subcutaneous injection, with some products available in prefilled pens 4.

Characteristics of GLP-1 Receptor Agonists

  • GLP-1 receptor agonists have a similar structure to human glucagon-like peptide-1 (GLP-1), but with modifications to increase their stability and half-life 4, 5.
  • These modifications can include the addition of a hydrophobic fatty acid side chain or a hydrophilic oligosaccharide 6.
  • GLP-1 receptor agonists can be classified as short-acting or long-acting, depending on their duration of action 7.

Research and Development

  • Researchers are actively exploring new GLP-1 receptor agonists with improved properties, such as increased potency and duration of action 8.
  • The development of small molecule GLP-1 receptor agonists is also an area of research, with potential advantages including lower cost and oral administration 8.
  • GLP-1 receptor agonists have shown promise in the treatment of type 2 diabetes and other diseases, and ongoing research aims to further optimize their design and therapeutic applications 7, 8.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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