Which statin (HMG-CoA reductase inhibitor) increases high-density lipoprotein (HDL) cholesterol?

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From the Guidelines

Rosuvastatin (Crestor) is the statin that most effectively increases HDL cholesterol levels, with typical increases of 5-10%. While all statins primarily work to lower LDL ("bad") cholesterol, rosuvastatin has demonstrated the greatest ability to raise HDL ("good") cholesterol. The standard dosage ranges from 5-40mg daily, with higher doses generally producing more significant HDL elevation. Atorvastatin (Lipitor) can also increase HDL, though typically to a lesser extent than rosuvastatin.

Key Points to Consider

  • The HDL-raising effect of statins occurs alongside their primary mechanism of inhibiting HMG-CoA reductase, which blocks cholesterol production in the liver.
  • This dual action makes rosuvastatin particularly valuable for patients with both elevated LDL and low HDL levels, as supported by studies such as 1.
  • Lifestyle modifications including regular exercise, weight management, smoking cessation, and moderate alcohol consumption can further enhance HDL levels when combined with statin therapy.
  • It's essential to note that while raising HDL is generally considered beneficial, the primary goal of statin therapy remains lowering LDL cholesterol to reduce cardiovascular risk, as emphasized in 1 and 1.

Additional Considerations

  • Other classes of drugs, such as fibrates and nicotinic acid, can also raise HDL-C levels, but their use may be limited by side effects and interactions with other medications, as discussed in 1 and 1.
  • The combination of a statin with nicotinic acid or a fibrate may produce a marked reduction of LDL-C and a striking rise in HDL-C, but this combination should be used with caution due to the potential risk of myopathy, as noted in 1.

From the FDA Drug Label

At one year, rosuvastatin increased HDL-C and reduced LDL-C, hsCRP, total cholesterol and serum triglyceride levels (p<0. 001 for all versus placebo). Primary Hyperlipidemia in Adults Rosuvastatin reduces Total-C, LDL-C, ApoB, non-HDL-C, and TG, and increases HDL-C, in adult patients with hyperlipidemia and mixed dyslipidemia

The statin (HMG-CoA reductase inhibitor) that increases high-density lipoprotein (HDL) cholesterol is rosuvastatin 2.

  • Key points:
    • Rosuvastatin increases HDL-C
    • Rosuvastatin reduces LDL-C, hsCRP, total cholesterol, and serum triglyceride levels
    • Rosuvastatin is effective in adult patients with hyperlipidemia and mixed dyslipidemia

From the Research

Statin Effects on HDL Cholesterol

  • Rosuvastatin has been found to increase high-density lipoprotein (HDL) cholesterol levels 3, 4, 5, 6, 7
  • The degree of HDL cholesterol increase with rosuvastatin is approximately 10% 4, which is greater than the increase seen with other statins (approximately 5%) 4
  • Rosuvastatin's ability to increase HDL cholesterol, in addition to its potent low-density lipoprotein (LDL) cholesterol reduction, makes it a valuable treatment option for patients with dyslipidemia 3, 5, 6, 7

Comparison to Other Statins

  • Rosuvastatin has been shown to have a greater efficacy in increasing HDL cholesterol compared to atorvastatin 6
  • Rosuvastatin's effects on HDL cholesterol are also favorable compared to other statins, such as simvastatin, pravastatin, and lovastatin 4, 5
  • The increased potency of rosuvastatin in lowering LDL cholesterol and increasing HDL cholesterol makes it a useful treatment option for patients who require aggressive lipid management 3, 4, 5, 6, 7

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Efficacy and safety of rosuvastatin in the management of dyslipidemia.

Vascular health and risk management, 2009

Research

Rosuvastatin in the management of hyperlipidemia.

Clinical therapeutics, 2004

Research

Efficacy and safety of rosuvastatin in treatment of dyslipidemia.

American journal of health-system pharmacy : AJHP : official journal of the American Society of Health-System Pharmacists, 2005

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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