What are the main functions of thyroid hormones?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: July 31, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Main Functions of Thyroid Hormones

Thyroid hormones are primary regulators of metabolic rate, controlling energy homeostasis through their effects on carbohydrate, lipid, and protein metabolism, as well as mitochondrial function and thermogenesis. 1

Metabolic Regulation

Thyroid hormones (T3 and T4) exert their physiologic actions through control of DNA transcription and protein synthesis. They diffuse into the cell nucleus and bind to thyroid receptor proteins attached to DNA, activating gene transcription and synthesis of messenger RNA and cytoplasmic proteins 2. The main metabolic functions include:

  • Basal Metabolic Rate: Thyroid hormones regulate substrate utilization, control basal metabolic rate, modulate heat production, and influence oxygen consumption 1
  • Carbohydrate Metabolism: Increased gluconeogenesis and enhanced glucose utilization 1
  • Lipid Metabolism: Enhanced cholesterol metabolism, bile acid synthesis, stimulation of lipogenesis and lipolysis 1
  • Protein Synthesis: Regulation of protein synthesis and degradation 1

Cardiovascular System Effects

Thyroid hormones have profound effects on the cardiovascular system:

  • Increase heart rate and contractility
  • Decrease systemic vascular resistance
  • Increase cardiac output (up to 300% from euthyroid state)
  • Enhance blood volume (up to 25% increase)
  • Stimulate erythropoietin release 1, 3

These effects explain why hyperthyroidism can lead to heart failure, particularly in patients over 50 years or with underlying cardiac disease 3, 1.

Growth and Development

Thyroid hormones are essential for:

  • Normal growth and skeletal development
  • Central nervous system development, particularly during fetal and early postnatal periods
  • Regulation of GH and IGF-1 effects on growth 3

Thyroid Hormone Physiology

The physiological actions of thyroid hormones are produced predominantly by T3, with approximately 80% of circulating T3 derived from peripheral conversion of T4 by deiodination 2. Key aspects of thyroid hormone physiology include:

  • Production: T4 and T3 are synthesized in the thyroid gland, with iodine as a crucial component 3
  • Transport: Circulating thyroid hormones are >99% bound to plasma proteins (TBG, TBPA, and albumin) 2
  • Metabolism: The liver is the major site of degradation for both T4 and T3, with T4 deiodination also occurring in kidney and other tissues 2
  • Elimination: Primarily through the kidneys, with approximately 20% eliminated in stool 2

Regulation of Thyroid Function

Thyroid hormone production is regulated by:

  • Hypothalamic-Pituitary-Thyroid Axis: TRH from hypothalamus stimulates TSH release from pituitary, which stimulates thyroid hormone production
  • Negative Feedback: T3 and T4 inhibit TRH and TSH production
  • Iodine Availability: Iodine is essential for thyroid hormone synthesis 3

Clinical Implications

Disruptions in thyroid hormone function lead to significant clinical consequences:

  • Hyperthyroidism: Increased metabolic rate, weight loss despite increased appetite, heat intolerance, tachycardia, and potential heart failure 3
  • Hypothyroidism: Decreased metabolic rate, weight gain, cold intolerance, bradycardia, and potential cognitive impairment 3

Important Considerations

  • Thyroid function testing is essential in patients with unexplained metabolic derangements 1
  • Selenium and iron are important cofactors for proper thyroid function 3
  • Thyroid disorders can significantly impact cardiovascular health, with both hypo- and hyperthyroidism associated with increased cardiovascular risk 4

Thyroid hormones represent a critical regulatory system affecting virtually every organ system in the body, with their primary role being the regulation of metabolic processes and energy homeostasis.

References

Guideline

Thyroid Hormones and Metabolic Regulation

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Thyroid hormone in health and disease.

The Journal of endocrinology, 2005

Related Questions

What is the recommended adjustment for a 32-year-old female patient with a TSH level of 3.53, Total T3 level of 1.45, and Free T3 level of 3.8, who is currently taking Levothyroxine (T4) 25 mcg daily?
Does a patient with a Thyroid-Stimulating Hormone (TSH) level of 1.59, Triiodothyronine (T3) level of 1.45, and Thyroxine (T4) level of 10.630 require thyroid hormone replacement therapy or antithyroid medication?
Does the risk of palpitations outweigh the risks of over-replacement with a TSH level of 0.030 in a patient on levothyroxine (thyroid hormone replacement medication)?
Can I resume the same dose of levothyroxine (thyroid hormone replacement medication) after being off of it for a week?
Should I initiate thyroxine (T4) therapy in a 65-year-old male patient with low free T4 levels, normal Thyroid-Stimulating Hormone (TSH) and Triiodothyronine (T3) levels, who is also taking escitalopram (Selective Serotonin Reuptake Inhibitor (SSRI)) 5mg?
For how long should nystatin (antifungal medication) swish and swallow be ordered for oral candidiasis?
What is pure seminoma?
Does a person born in 1962 need the measles, mumps, and rubella (MMR) vaccine?
Is a bladder biopsy done to check for complete response to BCG (Bacillus Calmette-Guérin) therapy in bladder cancer?
What is the diagnostic approach for adrenal insufficiency?
When to choose empagliflozin (Empagliflozin) over dapagliflozin (Dapagliflozin) and is their use recommended in patients with severe impaired renal function (eGFR below 15)?

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.