What are the types of hereditary hemochromatosis (HH)?

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From the Guidelines

Hereditary hemochromatosis (HH) is primarily classified into HFE-related and non-HFE-related types, with the most common forms being HFE-related, including C282Y/C282Y and C282Y/H63D genotypes, as well as non-HFE-related types such as hemojuvelin (HJV), transferrin receptor-2 (TfR2), ferroportin (SLC40A1), and hepcidin (HAMP) mutations 1. The classification of hereditary hemochromatosis can be broken down into several key categories, including:

  • HFE-related hemochromatosis, which includes genotypes such as C282Y/C282Y and C282Y/H63D
  • Non-HFE-related hemochromatosis, which includes mutations in genes such as:
    • Hemojuvelin (HJV)
    • Transferrin receptor-2 (TfR2)
    • Ferroportin (SLC40A1)
    • Hepcidin (HAMP) These forms of hemochromatosis are distinct from secondary iron overload, which can result from various conditions including iron-loading anemias, chronic liver disease, and parenteral iron overload 1. It's worth noting that the diagnosis and management of hemochromatosis have been outlined in practice guidelines, such as those published by the American Association for the Study of Liver Diseases in 2011 1, which provide a framework for understanding the different types of hereditary hemochromatosis and their underlying genetic causes. The most recent and highest quality study on this topic is the 2011 practice guideline by the American Association for the Study of Liver Diseases 1, which provides a comprehensive overview of the classification and management of hereditary hemochromatosis.

From the Research

Types of Hereditary Hemochromatosis (HH)

  • Classical HH is associated with mutations in the HFE gene, specifically C282Y homozygotes or C282Y/H63D compound heterozygotes 2, 3, 4, 5
  • Non-HFE associated HH is caused by mutations in other genes involved in iron metabolism, such as the SLC40A1 gene encoding the iron exporter ferroportin 6, 2
  • Juvenile and TFR2-related hemochromatoses are types of non-HFE hemochromatosis that show recessive inheritance, increased transferrin saturation, iron storage in hepatocytes, and responsiveness to phlebotomy 6
  • Ferroportin disease is a type of hemochromatosis caused by autosomal dominant mutations in the SLC40A1 gene, characterized by iron loading of Kupffer cells and normal transferrin saturation 6

Subgroups of Hemochromatosis

  • The first subgroup includes juvenile and TFR2-related hemochromatoses, which are due to hepcidin deficiency or the inability to increase hepcidin levels according to iron stores 6
  • The second subgroup is caused by autosomal dominant mutations in the SLC40A1 gene, leading to a hemochromatosis-like phenotype with dominant inheritance 6

Diagnosis and Treatment

  • The diagnosis of HH is established by genetic testing in patients with elevated transferrin saturation values 2
  • Patients with an established diagnosis of HH and iron overload should be treated with phlebotomy to achieve body iron depletion, followed by maintenance phlebotomy 2, 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Hereditary hemochromatosis.

Minerva medica, 2008

Research

Recognition and management of hereditary hemochromatosis.

American family physician, 2002

Research

Hereditary hemochromatosis.

Seminars in hematology, 2002

Research

Hereditary hemochromatosis: missed diagnosis or misdiagnosis?

The American journal of medicine, 2013

Research

Rare types of genetic hemochromatosis.

Acta haematologica, 2009

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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