How do I work up a patient with suspected dementia or Alzheimer's disease?

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Last updated: August 8, 2025View editorial policy

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Diagnostic Workup for Suspected Dementia or Alzheimer's Disease

A comprehensive diagnostic evaluation for suspected dementia should include a structured history, cognitive assessment, physical examination, laboratory testing, and brain imaging, followed by appropriate specialist referral for complex cases.

Initial Assessment

History Taking

  • Obtain detailed history of cognitive symptoms from both patient and reliable informant (family member/caregiver) 1, 2
  • Document specific examples of:
    • Memory difficulties (especially learning and recalling new information)
    • Changes in language abilities
    • Executive function problems (planning, organizing)
    • Visuospatial difficulties
    • Behavioral or personality changes
  • Establish timeline of symptom onset and progression 1
  • Assess impact on daily functioning and independence 1, 3

Cognitive Assessment

  • Administer validated cognitive screening tools:
    • Mini-Mental State Examination (MMSE) - sensitivity 88.3%, specificity 86.2% 2
    • Montreal Cognitive Assessment (MoCA) - more sensitive for mild cognitive impairment 2
    • Mini-Cog or GP Assessment of Cognition (GPCOG) for brief screening 2
  • Document baseline scores for future comparison 2
  • Consider educational level when interpreting results 2

Physical and Neurological Examination

  • Perform thorough neurological examination to identify:
    • Focal neurological signs (suggesting stroke or other structural lesions)
    • Extrapyramidal signs (suggesting Lewy body dementia or Parkinson's disease)
    • Gait abnormalities 1

Laboratory Testing

First-Tier Laboratory Tests (Essential)

  • Complete blood count
  • Comprehensive metabolic panel (including electrolytes, glucose, renal and liver function)
  • Thyroid function tests (TSH)
  • Vitamin B12 and folate levels
  • Homocysteine level 1, 2, 4

Second-Tier Laboratory Tests (Based on Clinical Suspicion)

  • Erythrocyte sedimentation rate (ESR)
  • C-reactive protein (CRP)
  • Syphilis serology (RPR/FTA-ABS)
  • HIV testing
  • Lyme disease antibodies
  • Antinuclear antibodies
  • Thyroid peroxidase antibodies (if autoimmune encephalopathy suspected) 1

Neuroimaging

Brain MRI (Preferred) or CT if MRI Contraindicated

  • Assess for:
    • Structural causes (tumors, hydrocephalus)
    • Vascular disease (infarcts, white matter changes)
    • Patterns of atrophy (medial temporal lobe atrophy in Alzheimer's disease) 1, 2, 3

Specialist Referral

When to Refer to a Specialist

  • Atypical cognitive presentations (aphasia, apraxia, agnosia)
  • Sensorimotor dysfunction
  • Severe mood/behavioral disturbances
  • Rapid progression or fluctuating course
  • Early age of onset (< 65 years)
  • Incongruent history and examination findings
  • Normal office-based cognitive testing despite concerning symptoms 1

Specialist Evaluation May Include

  • Detailed neurobehavioral assessment
  • Neuropsychological testing (when office-based assessment is insufficient) 1, 2
  • Advanced diagnostic tests:
    • CSF analysis for AD biomarkers (Aβ42, tau, phospho-tau)
    • FDG-PET scan
    • Amyloid PET scan 1, 2

Diagnostic Disclosure

  • Communicate findings honestly and compassionately to both patient and care partner 1
  • Provide information about:
    • Name and characteristics of the cognitive syndrome
    • Likely underlying disease(s)
    • Disease stage
    • Expected progression
    • Treatment options
    • Safety concerns
    • Available resources for education and support 1

Common Pitfalls to Avoid

  1. Attributing cognitive changes to "normal aging" - Cognitive decline that interferes with daily function is never normal aging 1, 3

  2. Missing treatable causes - Hypothyroidism, vitamin deficiencies, medication effects, and metabolic disorders can cause reversible cognitive impairment 4

  3. Overlooking depression - Pseudodementia due to depression can mimic dementia but is treatable 3, 5

  4. Inadequate cognitive testing - Brief office assessments may miss mild cognitive impairment; consider formal neuropsychological testing when clinical suspicion remains high despite normal screening 1, 2

  5. Failure to obtain collateral history - Patient self-report alone is insufficient; information from family/caregivers is essential 1

  6. Neglecting impact of medications - Review all medications for potential cognitive side effects; drug toxicity accounts for approximately 9.5% of dementia cases 4

By following this structured approach to evaluation, clinicians can accurately diagnose dementia, identify its underlying cause(s), and develop appropriate management plans to optimize patient outcomes.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Cognitive Assessment and Management of Progressive Cognitive Difficulties

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Alzheimer's disease: making the diagnosis.

American family physician, 1987

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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