ECG Criteria for Ventricular Tachycardia
The most reliable ECG criteria for diagnosing ventricular tachycardia (VT) include AV dissociation, fusion complexes, absence of RS complexes in precordial leads, and RS interval >100 ms in any precordial lead. 1, 2
Primary Diagnostic Criteria
AV Dissociation and Fusion Beats
- AV dissociation: Ventricular rate faster than atrial rate - pathognomonic for VT but only visible in about 30% of cases 1, 2
- Fusion complexes: Merger between conducted sinus impulses and ventricular depolarization - pathognomonic for VT 1, 2
- Physical exam signs of AV dissociation: Irregular cannon A waves in jugular venous pulse, variability in first heart sound loudness and systolic blood pressure 1
QRS Morphology (Brugada Criteria)
- Absence of RS complexes in all precordial leads (V1-V6) - indicates VT 1, 2
- RS interval >100 ms (onset of R wave to nadir of S wave) in any precordial lead - indicates VT 1, 2
QRS Width
- QRS width >140 ms with right bundle branch block (RBBB) pattern - suggests VT 1
- QRS width >160 ms with left bundle branch block (LBBB) pattern - suggests VT 1
- Note: QRS width alone is not definitive, as SVT with pre-existing BBB can have wide QRS 1
Additional Diagnostic Criteria
Vereckei Algorithm (aVR Lead)
- Initial R wave in aVR - suggests VT 1, 2
- Initial R or Q wave >40 ms in aVR - suggests VT 1, 2
- Notch on descending limb at onset of predominantly negative QRS - suggests VT 1, 2
Other Morphological Features
- QRS concordance: All positive or all negative QRS complexes across precordial leads - strongly suggests VT 1, 2
- R-wave peak time ≥50 ms in lead II - suggests VT 1, 2
- QR complex in one or more precordial leads V2-V6 - suggests VT 3
- Predominantly negative QRS complexes in leads V4-V6 - suggests VT 3
Algorithmic Approach to Wide Complex Tachycardia
- First step: Look for AV dissociation or fusion beats (pathognomonic for VT)
- Second step: Apply Brugada criteria (absence of RS complexes or RS interval >100 ms)
- Third step: Check QRS width (>140 ms with RBBB or >160 ms with LBBB)
- Fourth step: Examine aVR lead using Vereckei algorithm
- Fifth step: Look for QRS concordance across precordial leads
Important Clinical Considerations
- Do not rely on hemodynamic stability to distinguish SVT from VT - stable vital signs can occur in both conditions 1, 2
- When in doubt, treat as VT - especially before administering calcium channel blockers, which can cause hemodynamic collapse in VT 1
- QRS identical to sinus rhythm suggests SVT rather than VT 1
- SVT with pre-existing bundle branch block can be mistaken for VT 2
Pitfalls to Avoid
- Misdiagnosing VT as SVT with aberrancy: This can lead to inappropriate treatment with calcium channel blockers, potentially causing hemodynamic collapse 1
- Overreliance on single criteria: No single criterion is 100% sensitive or specific; use multiple criteria 4
- Ignoring clinical context: Patient history of structural heart disease increases likelihood of VT 3
- Failure to recognize fusion beats: These are subtle but pathognomonic for VT 1
By systematically applying these criteria, clinicians can accurately differentiate VT from SVT with aberrancy in most cases, leading to appropriate treatment decisions that reduce morbidity and mortality.