What is the average age of presentation for Neuropathy, Ataxia, and Retinitis Pigmentosa (NARP) mitochondrial disease?

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From the Research

NARP (Neuropathy, Ataxia, and Retinitis Pigmentosa) syndrome typically presents in childhood or early adulthood, with most patients developing symptoms between early childhood and young adulthood. The age of presentation can vary widely, with many cases becoming apparent in the first decade of life 1. The clinical manifestations often begin with developmental delay, learning disabilities, or seizures in childhood, followed by progressive symptoms including peripheral neuropathy, ataxia (loss of coordination), and retinitis pigmentosa (vision loss) 2. Some patients may present with more severe manifestations in infancy, including failure to thrive, while others might have milder symptoms that don't become apparent until adulthood 3. The variability in age of presentation relates to heteroplasmy (the proportion of mutated mitochondrial DNA), with higher mutation loads generally correlating with earlier and more severe disease 4. Key points to consider in the diagnosis and management of NARP syndrome include:

  • Maternal inheritance patterns are typical, with affected mothers transmitting the mutation to all their children, though the severity can differ significantly even within families due to varying levels of heteroplasmy 5
  • A genetic etiology should be established to confirm the diagnosis, and molecular-genetic methods are usually used for this purpose 1
  • Symptomatic treatment can improve patients' quality of life, and a correct diagnosis is the prerequisite for genetic counseling and personalized treatment 2

References

Research

Mitochondrial Disorders.

Deutsches Arzteblatt international, 2021

Research

Clinical manifestation of mitochondrial diseases.

Developmental period medicine, 2015

Research

Mitochondrial DNA alterations and genetic diseases: a review.

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 1994

Research

Molecular genetic testing for mitochondrial disease: from one generation to the next.

Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics, 2013

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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