What is Common Variable Immunodeficiency (CVID)?

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Common Variable Immunodeficiency (CVID)

Common Variable Immunodeficiency (CVID) is a primary immunodeficiency characterized by low immunoglobulin levels, impaired antibody production, and increased susceptibility to recurrent infections, with additional complications including autoimmune disorders, gastrointestinal disease, and malignancies. 1, 2

Definition and Diagnostic Criteria

CVID is defined by:

  • Low serum IgG levels (below normal range for age)
  • Low IgA and/or IgM levels
  • Impaired specific antibody production to vaccines or infections
  • Exclusion of other causes of hypogammaglobulinemia
  • Age >4 years (diagnosis not appropriate before this age) 1
  • Normal or reduced B cell numbers in peripheral blood 1

Laboratory findings typically show:

  • Reduced levels of at least 2 immunoglobulin isotypes
  • Poor antibody response to vaccines (particularly polysaccharide antigens)
  • B-cell abnormalities on flow cytometry 2

Epidemiology

  • Prevalence: 1 in 25,000 to 1 in 50,000 individuals worldwide 3
  • Most common symptomatic primary antibody deficiency 4
  • Typically presents in young adulthood (20s-30s), but can occur at any age 3
  • Only 10% of cases have an identified genetic cause 3

Clinical Manifestations

Infectious Complications

  • Recurrent sinopulmonary infections (most common presentation)
  • Bronchitis and pneumonia (can lead to bronchiectasis in 10-20% of patients) 1
  • Otitis media
  • Gastrointestinal infections (Giardiasis, Campylobacter jejuni, Salmonella) 1
  • Viral enteritis (CMV, norovirus, parechovirus) 1

Non-infectious Complications

  1. Pulmonary:

    • Bronchiectasis (10-20% of patients)
    • Granulomatous and lymphocytic interstitial lung disease (GLILD) (10% of patients)
    • Asthma-like presentation (10-15% of patients) 1
  2. Gastrointestinal (20-25% of patients):

    • Chronic gastritis
    • Pernicious anemia
    • Lymphoid nodular hyperplasia
    • Villous atrophy
    • Inflammatory bowel disease
    • Enteropathy 1
  3. Autoimmune disorders (20% of patients):

    • Autoimmune cytopenias (most common: thrombocytopenia, hemolytic anemia) (11-12%)
    • Arthritis
    • Psoriasis
    • Vitiligo 1, 5
  4. Liver abnormalities (40% of patients):

    • Elevated alkaline phosphatase
    • Nodular regenerative hyperplasia (can lead to portal hypertension) 1
  5. Lymphoproliferative disorders:

    • Lymphadenopathy
    • Splenomegaly
    • Increased risk of lymphoma 1

Classification

B-cell subset analysis by flow cytometry can help classify CVID patients (EUROclass):

  • Decreased switched memory B cells correlates with granulomatous disease and splenomegaly
  • Expansion of CD21low B cells correlates with splenomegaly and autoimmunity
  • Expansion of transitional B cells correlates with lymphadenopathy 1

Management

Immunoglobulin Replacement Therapy

  • Primary treatment: IgG replacement therapy (400-600 mg/kg every 3-4 weeks)
  • Higher doses (600 mg/kg/month) for patients with bronchiectasis or pulmonary complications 2
  • Available as intravenous (IVIG) or subcutaneous (SCIG) formulations 2
  • Regular monitoring of IgG trough levels every 6-12 months 2

Antimicrobial Therapy

  • Aggressive antimicrobial treatment for infections
  • Prophylactic antibiotics for patients with recurrent infections despite adequate IgG replacement 1

Monitoring and Follow-up

  • Regular pulmonary function testing
  • Monitoring of liver function tests
  • Gastrointestinal evaluation when symptoms present
  • Vigilance for autoimmune diseases and malignancies 1, 2

Special Considerations

  • Good syndrome: CVID with thymoma requires thymoma excision 1
  • Stem cell transplantation: May be considered for patients with malignancy or severe organ damage 1
  • Joint care: Patients with bronchiectasis should be under joint care of immunologist and respiratory specialist 2

Prognosis

Prognosis depends on:

  • Early diagnosis and treatment
  • Presence and severity of complications
  • Non-infectious complications (particularly GLILD, enteropathy, and malignancy) associated with increased mortality 1

Early diagnosis and prompt initiation of immunoglobulin replacement therapy are crucial to prevent permanent organ damage and improve quality of life 2.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Immunodeficiency Treatment Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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