What is the definition of secondary hypertension?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: August 24, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Definition of Secondary Hypertension

Secondary hypertension is defined as high blood pressure caused by an identifiable and potentially correctable underlying condition or disease, present in approximately 10% of adult patients with hypertension. 1

Key Characteristics

Secondary hypertension differs from primary (essential) hypertension in several important ways:

  • It has a specific identifiable cause (unlike primary hypertension where the cause is unknown)
  • The underlying condition may be potentially treatable or even curable
  • It accounts for approximately 5-10% of all hypertension cases in adults 1, 2
  • In children, secondary causes are more common, especially in those younger than 6 years 3

Common Causes of Secondary Hypertension

The most frequent causes include:

  • Obstructive sleep apnea (most prevalent secondary cause, present in up to 83% of patients with resistant hypertension) 4
  • Primary aldosteronism (present in approximately 20% of resistant hypertension cases) 4
  • Renovascular disease (renal artery stenosis)
  • Renal parenchymal disease
  • Drug and substance use (NSAIDs, oral contraceptives, corticosteroids, decongestants, anticancer drugs, cocaine, amphetamines) 4
  • Endocrine disorders (pheochromocytoma, Cushing syndrome, thyroid and parathyroid conditions)
  • Congenital conditions (coarctation of aorta, congenital adrenal hyperplasia) 1

Clinical Features Suggesting Secondary Hypertension

Certain clinical features should prompt evaluation for secondary causes:

  • Onset of hypertension before age 20 or after age 50 5
  • Sudden worsening of previously controlled hypertension
  • Malignant hypertension with end-organ damage (BP ≥180/110 mmHg with acute hypertension-mediated organ damage) 4
  • Resistant hypertension (requiring more than 3 medications from different classes) 5
  • Specific physical findings (abdominal bruits, sudden rise in creatinine with ACE inhibitors) 4, 5
  • Unprovoked hypokalemia (suggesting primary aldosteronism or renovascular disease) 5
  • Characteristic body habitus (truncal obesity, purple striae in Cushing's syndrome) 5

Diagnostic Approach

The European Heart Journal recommends the following diagnostic tests for suspected secondary hypertension 4:

Suspected Cause Recommended Screening Test
Primary aldosteronism Aldosterone-to-renin ratio
Renovascular hypertension Renal Doppler ultrasound, CT/MR angiography
Pheochromocytoma 24h urinary/plasma metanephrines and normetanephrines
Obstructive sleep apnea Overnight polysomnography
Renal parenchymal disease Renal ultrasound, urinalysis, eGFR
Cushing's syndrome 24h urinary free cortisol, dexamethasone suppression
Thyroid disease TSH
Hyperparathyroidism PTH, calcium, phosphate
Coarctation of aorta Echocardiogram, CT angiogram

Clinical Significance

Early recognition of secondary hypertension is important because:

  • It can lead to more effective, targeted treatment approaches 1
  • Some causes may be potentially curable with specific interventions
  • Untreated secondary causes can lead to accelerated target organ damage 2
  • Even with treatment of the underlying cause, blood pressure may not return to normal due to vascular remodeling, highlighting the importance of early detection 2

Management Principles

Management involves treating the underlying condition, which may include:

  • Primary aldosteronism: Unilateral adrenalectomy for unilateral disease or mineralocorticoid receptor antagonists for bilateral disease 4
  • Renovascular disease: Renal angioplasty for fibromuscular dysplasia; optimal cardiovascular risk management with consideration of stenting for atherosclerotic disease 4
  • Obstructive sleep apnea: Weight loss, CPAP therapy, mandibular advancement devices 4
  • Pheochromocytoma: Surgical removal after adequate alpha-blockade (alpha-blockers must be initiated before beta-blockers) 4
  • Medication-induced: Discontinuation or substitution of causative agents when possible

Important Clinical Considerations

  • Despite treatment of the underlying cause, hypertension may persist due to concomitant essential hypertension or irreversible vascular changes 2
  • Ambulatory blood pressure monitoring is essential to rule out white-coat hypertension and confirm true resistant hypertension 4
  • In children, secondary causes are significantly more common in those younger than 6 years, while 85-95% of hypertension in adolescents is primary 3
  • Referral to specialists (endocrinology, nephrology, vascular surgery) should be considered for complex cases 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Hypertension Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Secondary hypertension: evaluation and treatment.

Disease-a-month : DM, 1996

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.