What receptors does aripiprazole interact with?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: September 6, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Aripiprazole's Receptor Interaction Profile

Aripiprazole primarily acts as a partial agonist at dopamine D2 and D3 receptors, a partial agonist at serotonin 5-HT1A receptors, and an antagonist at 5-HT2A receptors, with additional moderate affinity for several other receptors. 1

Primary Receptor Interactions

Aripiprazole exhibits high binding affinity for:

  • Dopamine receptors:

    • D2 receptors (Ki = 0.34 nM) - partial agonist activity
    • D3 receptors (Ki = 0.8 nM) - partial agonist activity
  • Serotonin receptors:

    • 5-HT1A receptors (Ki = 1.7 nM) - partial agonist activity
    • 5-HT2A receptors (Ki = 3.4 nM) - antagonist activity

Secondary Receptor Interactions

Aripiprazole shows moderate affinity for:

  • Dopamine D4 receptors (Ki = 44 nM)
  • Serotonin 5-HT2C receptors (Ki = 15 nM)
  • Serotonin 5-HT7 receptors (Ki = 39 nM)
  • Alpha-1 adrenergic receptors (Ki = 57 nM)
  • Histamine H1 receptors (Ki = 61 nM)
  • Serotonin reuptake site (Ki = 98 nM)

Receptors with No Significant Interaction

Aripiprazole has no appreciable affinity for:

  • Cholinergic muscarinic receptors (IC50 > 1000 nM) 1

Clinical Significance of Receptor Profile

The unique receptor binding profile of aripiprazole contributes to its clinical effects and side effect profile:

  • Partial D2 agonism: Unlike traditional antipsychotics that are D2 antagonists, aripiprazole's partial agonist activity at D2 receptors allows it to act as a functional antagonist in dopamine-rich environments and as a functional agonist in dopamine-deficient environments 2

  • 5-HT1A partial agonism and 5-HT2A antagonism: This combination may contribute to improvements in negative symptoms and reduced extrapyramidal side effects 3

  • Low H1 and muscarinic receptor affinity: Results in minimal sedation and anticholinergic side effects compared to other antipsychotics 4

PET Study Findings on Receptor Occupancy

A triple tracer PET study in patients with schizophrenia showed:

  • Very high occupancy at striatal D2 receptors (87-93%)
  • Moderate occupancy at 5-HT2A receptors (54-60%)
  • Low occupancy at 5-HT1A receptors (16%) 3

This unique occupancy profile distinguishes aripiprazole from other antipsychotics and may explain its favorable side effect profile despite high D2 receptor occupancy.

Metabolite Activity

Aripiprazole's major metabolite, dehydro-aripiprazole:

  • Has similar affinity for D2 receptors as the parent compound
  • Represents approximately 40% of parent drug exposure in plasma
  • Has a longer elimination half-life (94 hours vs. 75 hours for aripiprazole) 1

The combination of aripiprazole's unique receptor profile and its active metabolite contributes to its overall clinical efficacy and tolerability in various psychiatric conditions.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.