What is the half-life of Crestor (rosuvastatin)?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: September 8, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Half-Life of Rosuvastatin (Crestor)

Rosuvastatin has a half-life of approximately 19 hours, which is among the longest half-lives of all statin medications. 1

Pharmacokinetic Properties of Rosuvastatin

Rosuvastatin has several key pharmacokinetic characteristics that distinguish it from other statins:

  • Elimination half-life: 19 hours 1
  • Absorption: Peak plasma concentrations reached 3-5 hours after oral dosing 1
  • Bioavailability: Approximately 20% 1
  • Distribution: Mean volume of distribution at steady-state is approximately 134 liters 1
  • Protein binding: 88% bound to plasma proteins, mostly albumin 1
  • Metabolism: Not extensively metabolized; only about 10% of a dose is recovered as metabolite 1
  • Excretion: Primarily excreted in feces (90%) 1

Comparison to Other Statins

Rosuvastatin's long half-life provides several clinical advantages:

Statin Half-life (hours)
Rosuvastatin 19
Atorvastatin 14
Pitavastatin 12
Fluvastatin 3
Simvastatin 2-3
Pravastatin 1.8

2

Clinical Implications of Rosuvastatin's Long Half-Life

The extended half-life of rosuvastatin offers several clinical benefits:

  • Sustained lipid-lowering effect: The long half-life contributes to rosuvastatin's potent and sustained lipid-lowering effects 3
  • Once-daily dosing: The pharmacokinetic profile supports convenient once-daily administration 1
  • Alternative dosing regimens: The long half-life makes rosuvastatin suitable for alternate-day dosing in patients who experience statin-related side effects with daily administration 4
  • Minimal drug interactions: Rosuvastatin undergoes minimal metabolism via the cytochrome P450 system, reducing the potential for drug interactions 3

Special Population Considerations

  • Renal impairment: Plasma concentrations increase approximately 3-fold in patients with severe renal impairment (CLcr < 30 mL/min/1.73 m²) 1
  • Asian patients: Asian subjects show approximately 2-fold elevation in median exposure (AUC and Cmax) compared to White subjects, requiring dosage adjustment 1
  • Elderly patients: No significant differences in plasma concentrations between elderly (≥65 years) and non-elderly populations 1

Clinical Relevance

The long half-life of rosuvastatin contributes to its high potency and efficacy in reducing LDL cholesterol (52-63% reduction with 10-40 mg doses) 3. This pharmacokinetic property allows for consistent lipid-lowering effects throughout the 24-hour dosing interval, potentially improving patient adherence and clinical outcomes.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.