What is the chemical mediator in a scald burn?

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Chemical Mediator in Spilled Hot Water Burns

Histamine is the primary chemical mediator in a scald burn, triggering the immediate inflammatory cascade after hot water exposure. 1

Pathophysiology of Scald Burns

The inflammatory response in thermal injuries follows a specific sequence:

  1. Initial phase: Histamine is released as the earliest chemical mediator following thermal injury 1, 2

    • Causes immediate vasodilation and increased microvascular permeability
    • Triggers the initial inflammatory cascade
  2. Secondary mediators: Following histamine release, other mediators are activated in sequence 1, 3:

    • Bradykinins
    • Prostaglandins
    • Thromboxanes
    • Leukotrienes

Mechanism of Action

Histamine's role in scald burns involves:

  • Direct activation of vascular endothelium causing increased permeability 2
  • Creation of endothelial gaps allowing fluid shift into interstitial spaces
  • Triggering of the earliest stage of vasodilation 2

The inflammatory cascade progresses with a "two-hit" model rather than a simple linear pathway:

  • Histamine acts as the vascular permeability-increasing mediator
  • Prostaglandins subsequently act as vasodilators that potentiate plasma exudation 4

Clinical Implications

Understanding histamine as the primary mediator has important treatment implications:

  • Immediate cooling of burns helps limit the histamine-mediated inflammatory response 1
  • Fluid management is critical as histamine-triggered increases in vascular permeability lead to significant fluid shifts 1, 3
  • Pain management requires multimodal approaches as the histamine-initiated cascade contributes significantly to burn pain 1

Common Pitfalls

  • Misattribution: While prostaglandins, thromboxanes, and bradykinins are important in burn pathophysiology, they are not the initial mediators 1, 2
  • Oversimplification: The inflammatory response is complex and involves multiple mediators working in sequence and synergistically 4
  • Treatment delay: Failure to recognize histamine's early role may delay appropriate cooling interventions that could limit the inflammatory cascade 1

Therefore, among the options provided (Serotonin, Bradykinins, Thromboxane, Prostaglandins), none represent the primary chemical mediator in a spilled hot water burn. The correct answer would be Histamine.

References

Guideline

Burn Pathophysiology and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Pathophysiology of the burn wound.

Annales chirurgiae et gynaecologiae, 1980

Research

[Burn shock, diagnostics, monitoring and fluid therapy of severe burns--new look].

Wiadomosci lekarskie (Warsaw, Poland : 1960), 2011

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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