Pain Mechanisms in Gluteus Minimus/Anterior Medius Insertional Tendinosis
Gluteus minimus/anterior medius insertional tendinosis causes pain primarily through a combination of excessive compression and high tensile loads at the tendon insertion site, leading to degenerative changes and inflammatory responses. 1
Pathophysiological Mechanisms of Pain
The pain experienced in gluteus minimus/anterior medius insertional tendinosis stems from several key mechanisms:
Mechanical Factors:
- Excessive compression at the greater trochanter insertion site
- High tensile loads during hip movement, particularly with hip adduction 1
- Abnormal biomechanics causing repeated microtrauma to the tendon
Structural Changes:
- Tendinous enlargement and heterogeneous tissue structure
- Degenerative changes in the tendon rather than acute inflammation 2
- Possible development of partial-thickness tears within the tendon substance
Neurological Factors:
- Sensitization of nociceptors in the affected tendon
- Increased innervation in areas of tendinosis
- Pain during both loading activities and rest (particularly side-lying) 1
Clinical Presentation and Assessment
Gluteus minimus/anterior medius insertional tendinosis typically presents with:
- Lateral hip pain that worsens with activity and side-lying 1
- Weakness in hip abduction 3
- Possible positive Trendelenburg sign and altered gait in more advanced cases 3
Diagnosis is confirmed through:
- Ultrasound or MRI showing tendinous changes without complete rupture 3
- Clinical tests that provoke both compressive and tensile loads on the affected tendons 1
Management Considerations
Treatment should follow a progressive approach:
Initial Management:
Intermediate Management:
Advanced Management:
Common Pitfalls and Considerations
- Misdiagnosis as trochanteric bursitis is common; proper imaging is essential
- Excessive cortisone injections can weaken tendon structure; limit to 2-3 injections with 4-6 weeks between them 5
- Failure to address biomechanical factors (especially excessive hip adduction) may lead to persistent symptoms 1
- Surgical repair should be reserved for cases with failed conservative management or significant functional limitations 4
Rehabilitation should focus on gradually increasing load tolerance while avoiding positions that create excessive compression at the greater trochanter, particularly during the early phases of treatment 4.