What are the types of shock and how are they assessed using the look, listen, feel approach?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: September 18, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Types of Shock: Assessment Using Look, Listen, Feel Approach

Shock is classified into four major categories based on pathophysiology: hypovolemic, distributive, cardiogenic, and obstructive shock, each requiring specific management approaches tailored to the underlying mechanism. 1

Classification of Shock Types

1. Hypovolemic Shock

  • Definition: Results from intravascular volume loss
  • Causes: Hemorrhage, severe dehydration, excessive diuresis, vomiting, diarrhea 1
  • Look:
    • Tachycardia
    • Weak pulses
    • Cold, pale extremities
    • Decreased skin turgor
    • Dry mucous membranes
  • Listen:
    • Rapid, thready pulse
    • Clear lung fields
  • Feel:
    • Capillary refill time >2 seconds
    • Cool skin
    • Weak peripheral pulses

2. Distributive Shock

  • Definition: State of relative hypovolemia from pathological redistribution of intravascular volume
  • Types: Septic shock, anaphylactic shock, neurogenic shock, adrenal crisis 1
  • Look:
    • Warm, flushed skin (early septic shock)
    • Urticaria/angioedema (anaphylactic)
    • Hypotension
    • Altered mental status
  • Listen:
    • Bounding pulses
    • Possible wheezing (anaphylactic)
    • Possible crackles (septic)
  • Feel:
    • Warm peripheries (early)
    • Fever (septic)
    • Rapid pulse

3. Cardiogenic Shock

  • Definition: Inadequate cardiac output due to primary cardiac dysfunction
  • Causes: Myocardial infarction, heart failure, valvular disease, arrhythmias, cardiomyopathy 1
  • Look:
    • Hypotension (SBP <90 mmHg for >30 minutes)
    • Jugular venous distention
    • Peripheral edema
    • Cyanosis
  • Listen:
    • S3 gallop
    • Pulmonary crackles
    • Murmurs (valvular causes)
  • Feel:
    • Cold, clammy skin
    • Weak pulses
    • Pulmonary edema

4. Obstructive Shock

  • Definition: Mechanical obstruction to blood flow causing decreased cardiac output
  • Causes: Pulmonary embolism, tension pneumothorax, cardiac tamponade, constrictive pericarditis 1
  • Look:
    • Hypotension
    • Elevated jugular venous pressure
    • Unilateral chest expansion (tension pneumothorax)
    • Cyanosis
  • Listen:
    • Muffled heart sounds (tamponade)
    • Unilateral decreased breath sounds (pneumothorax)
    • Tracheal deviation (tension pneumothorax)
  • Feel:
    • Pulsus paradoxus (tamponade)
    • Tracheal deviation (tension pneumothorax)
    • Subcutaneous emphysema (pneumothorax)

Diagnostic Assessment

Vital Signs Assessment

  • Blood pressure: Hypotension (SBP <90 mmHg) is common to all shock types 1
  • Heart rate: Typically elevated (except in neurogenic shock)
  • Respiratory rate: Usually increased
  • Temperature: Elevated in septic shock, may be decreased in late shock states
  • Oxygen saturation: Often decreased

Hemodynamic Parameters

  • Cardiac index: Target ≥2.2 L/min/m² 1
  • Mixed venous oxygen saturation: Target ≥70% 1
  • Mean arterial pressure: Target ≥70 mmHg 1
  • Urine output: Target >30 mL/h 1

Laboratory Assessment

  • Lactate levels: Elevated (>2 mmol/L) indicates tissue hypoperfusion 1
  • Complete blood count: May show anemia (hypovolemic) or leukocytosis (septic)
  • Coagulation studies: May reveal DIC in septic shock
  • Cardiac biomarkers: Elevated in cardiogenic shock
  • Blood cultures: For suspected septic shock

Management Approach Based on Shock Type

Hypovolemic Shock

  • Fluid resuscitation with balanced crystalloids as first-line treatment 1
  • Blood products for hemorrhagic shock
  • Address underlying cause (control bleeding, replace fluid losses)

Distributive Shock

  • Combination of vasopressors and fluid replacement 1
  • Norepinephrine as first-line vasopressor after adequate fluid resuscitation
  • Specific treatments for underlying cause (antibiotics for sepsis, epinephrine for anaphylaxis)

Cardiogenic Shock

  • Inotropic support and addressing underlying cause 1
  • Dobutamine as first-line inotrope (2-20 μg/kg/min) for acute heart failure
  • Consider mechanical circulatory support based on failure pattern:
    • Left ventricular failure: Impella devices, IABP, or TandemHeart
    • Right ventricular failure: Impella RP or TandemHeart Protek-Duo
    • Biventricular failure: Bilateral Impella pumps or VA-ECMO with LV venting 1

Obstructive Shock

  • Immediate life-saving intervention to relieve obstruction 1
    • Needle decompression for tension pneumothorax
    • Pericardiocentesis for cardiac tamponade
    • Anticoagulation, thrombolysis, or embolectomy for pulmonary embolism

Special Considerations

Ventilatory Support

  • Consider early endotracheal intubation and mechanical ventilation to reduce work of breathing and improve oxygenation 1
  • Non-invasive positive pressure ventilation may be considered for respiratory distress in non-intubated patients 1

Multidisciplinary Care

  • A multidisciplinary shock team approach has been associated with improved 30-day all-cause mortality (HR, 0.61; 95% CI, 0.41–0.93) in patients with cardiogenic shock 1

Common Pitfalls and Caveats

  1. Misdiagnosis of shock type: Incorrect identification can lead to inappropriate treatment. Use the look, listen, feel approach systematically.

  2. Delayed recognition of pre-shock: Patients with compensatory vasoconstriction may maintain near-normal systolic blood pressure despite malperfusion, which can be falsely reassuring 2

  3. Failure to recognize mixed shock states: Patients may present with features of multiple shock types simultaneously.

  4. Over-reliance on single parameters: Integrate clinical, laboratory, and hemodynamic data for accurate diagnosis.

  5. Inadequate monitoring: Continuous reassessment is essential as shock states can evolve rapidly.

  6. Delayed intervention: Prompt recognition and treatment are crucial for improving outcomes.

References

Guideline

Shock Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.