What is the percentage chance that a Proton Pump Inhibitor (PPI) will cause a kidney stone?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: September 18, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Risk of Kidney Stone Formation with Proton Pump Inhibitors

Based on the most recent and highest quality evidence, proton pump inhibitors (PPIs) are associated with approximately a 19-46% increased risk of kidney stone formation, with the absolute risk being relatively small.

Evidence on PPI Use and Kidney Stone Risk

Recent research demonstrates a clear association between PPI use and kidney stone formation:

  • A large retrospective study found that PPI use was associated with a 46% increased risk of kidney stones (HR 1.46; 95% CI 1.38-1.55) 1
  • Another study of patients with GERD showed a 19% higher risk of incident kidney stones with PPI use (HR 1.19; 95% CI 1.06-1.34) 2
  • Meta-analysis data indicates an overall hazard ratio of 1.34 (95% CI 1.28-1.40) for incident nephrolithiasis with PPI use 3
  • The risk appears to be dose-dependent, with higher doses associated with greater risk (HR 1.11; CI 1.09-1.14 for each increase in 30 defined daily doses over a 3-month period) 1

Mechanisms of PPI-Associated Kidney Stone Formation

The increased risk of kidney stones with PPI use appears to be related to several mechanisms:

  • PPIs are associated with decreased urinary citrate excretion (12% lower 24-hour urine citrate) 4, which is an important inhibitor of stone formation
  • PPI-exposed patients have significantly lower urinary magnesium levels (mean 3.6 vs 4.3 mmol, p < 0.001) 2, another inhibitor of stone formation
  • The risk increases with duration of exposure, becoming apparent after three months of PPI use (HR 1.78; 95% CI 1.39-2.25) 5

Clinical Implications and Recommendations

Despite these findings, current gastroenterology guidelines do not recommend avoiding PPIs solely due to kidney stone risk. The American Gastroenterological Association (AGA) states that "the decision to discontinue PPIs should be based solely on the lack of an indication for PPI use, and not because of concern for PPI-associated adverse events" 6.

When prescribing PPIs:

  • Use the lowest effective dose and shortest duration possible to minimize risks 7
  • Consider monitoring urinary citrate and magnesium levels in patients with history of kidney stones who require long-term PPI therapy
  • Be aware that the risk appears to be dose-dependent and increases with longer duration of use
  • H2-receptor antagonists may also increase kidney stone risk (HR 1.47; CI 1.31-1.64) 1, suggesting acid suppression itself may be an involved mechanism

Conclusion

While PPIs are associated with a modest increased risk of kidney stones (approximately 19-46% relative risk increase), the absolute risk remains small for most patients. This risk should be considered in the context of the clear benefits of PPI therapy for appropriate indications.

References

Research

Use of Proton Pump Inhibitors Increases Risk of Incident Kidney Stones.

Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association, 2021

Research

Risk of urinary stone formation associated to proton pump inhibitors: A systematic review and metanalysis.

Archivio italiano di urologia, andrologia : organo ufficiale [di] Societa italiana di ecografia urologica e nefrologica, 2022

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Proton Pump Inhibitors and Electrolyte Disturbances

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.