What process is likely deficient in insulin missing the C peptide (Connecting peptide)?

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Deficient Process in Recombinant Insulin Missing C-peptide

The early forms of recombinant insulin produced in bacteria that were missing the C-peptide were most likely to be deficient in the stability of the fully folded protein (option D).

Understanding C-peptide's Role in Insulin Structure and Function

The C-peptide (connecting peptide) plays a crucial role in insulin biosynthesis and protein stability:

  • In normal insulin production, preproinsulin is synthesized and translocated into the endoplasmic reticulum where the signal peptide is cleaved to form proinsulin 1
  • Proinsulin consists of the insulin B-chain, C-peptide, and A-chain in a continuous single polypeptide 2
  • The C-peptide facilitates proper folding of proinsulin and formation of the three disulfide bonds critical for insulin's structure 1

Why C-peptide Absence Affects Protein Stability

  • The C-peptide serves as a molecular chaperone that ensures proper spatial alignment of the A and B chains during folding 3
  • Without the C-peptide, recombinant insulin has reduced stability of the fully folded protein structure 3
  • Studies show that constructs with shortened or absent C-peptide linkers tend to be synthesized at lower levels and exhibit improper disulfide bonding 3

Evidence Against Other Options

  • A. Binding to insulin receptor by fully folded protein: The binding of insulin to its receptor is primarily determined by specific amino acid residues in the A and B chains, not by the C-peptide which is cleaved before secretion 4

  • B. Rapid formation of the appropriate S-S bonds: While C-peptide does assist in disulfide bond formation, this is a secondary effect of its primary role in ensuring proper spatial alignment for stable folding 3

  • C. Release from secretory granules: C-peptide is cleaved from proinsulin in secretory granules, but its absence in recombinant insulin wouldn't affect release mechanisms since bacterial expression systems don't use secretory granules 4

  • E. Translation of the A and B fragments: Translation itself isn't affected by C-peptide absence; rather, it's the post-translational folding and stability that are compromised 2

Clinical Relevance of C-peptide

  • C-peptide is secreted equimolarly with insulin into circulation and is used clinically to assess endogenous insulin production 4
  • Low or undetectable levels of plasma C-peptide are indicative of little or no insulin secretion, as seen in advanced type 1 diabetes 5
  • C-peptide measurements help distinguish between type 1 and type 2 diabetes in ambiguous cases 5
  • Beyond its role in insulin biosynthesis, C-peptide has been found to have biological activities including binding to cell membranes and triggering intracellular signaling 6

In conclusion, the absence of C-peptide in early recombinant insulin primarily affected the stability of the fully folded protein, leading to challenges in producing properly structured and functional insulin molecules.

References

Research

Role of the connecting peptide in insulin biosynthesis.

The Journal of biological chemistry, 2003

Research

C-peptide.

Diabetes care, 1982

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

N-terminal segment of proinsulin C-peptide active in insulin interaction/desaggregation.

Biochemical and biophysical research communications, 2010

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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