What are the side effects of amiodarone?

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Side Effects of Amiodarone

Amiodarone has numerous potentially harmful side effects affecting multiple organ systems, with pulmonary toxicity being the most serious adverse effect requiring regular monitoring and prompt intervention. 1

Major Organ System Effects

Pulmonary Toxicity

  • Most serious potential adverse effect of amiodarone therapy 1
  • Presents as subacute cough and progressive dyspnea with patchy interstitial infiltrates on chest radiographs 1
  • Reduced diffusing capacity on pulmonary function tests 1
  • Any report of worsening dyspnea or cough should prompt immediate assessment 1
  • Treatment involves withdrawal of amiodarone, supportive care, and sometimes corticosteroids 1

Thyroid Dysfunction

  • Occurs in 2-24% of patients receiving long-term amiodarone therapy 1
  • Can cause both hyperthyroidism (from excess iodine or acute thyroiditis) and hypothyroidism (2-4 times more common than hyperthyroidism) 1, 2
  • Regular monitoring of thyroid function tests is mandatory 1
  • In hypothyroid patients with strong indication for amiodarone, the drug may be continued with thyroid hormone supplementation 1

Liver Toxicity

  • Manifested by elevation of liver transaminase levels 1
  • Occurs at a rate of 0.6% annually 1
  • Patients are rarely symptomatic 1
  • If liver enzyme levels exceed three times normal, amiodarone should be discontinued unless patient is at high risk for recurrence of life-threatening arrhythmia 1

Cardiovascular Adverse Effects

  • Bradycardia and heart block occur in 1-3% of patients 1
  • Amiodarone-induced proarrhythmia occurs at an annual rate of <1% 1
  • QT interval prolongation is common, but torsades de pointes is rare 1
  • Contraindicated in patients with second- or third-degree heart block without a pacemaker 1
  • IV administration can cause hypotension (16% of patients) 1

Other Common Side Effects

Neurologic Toxicity

  • Includes ataxia, paresthesias, and tremor 1
  • Often dosage-related and improve with dose reduction 1
  • Peripheral neuropathy occurs at a rate of 0.3% annually 1

Dermatologic Adverse Effects

  • Photosensitivity is common - patients should use sunblock and cover exposed skin outdoors 1
  • Bluish skin discoloration may develop in areas with extended sun exposure 1
  • Resolves over several months after discontinuation 1

Ocular Adverse Effects

  • Corneal microdeposits visible on slit-lamp examination in nearly all patients 1
  • Rarely affect vision or require discontinuation 1
  • Optic neuropathy and optic neuritis have been reported in rare cases 1
  • Any changes in visual acuity or peripheral vision warrant ophthalmologic evaluation 1

Gastrointestinal Adverse Effects

  • Include nausea, anorexia, and constipation 1
  • Often dosage-related and improve with dose reduction 1

Monitoring Recommendations

Baseline Assessment

  • Complete history and physical examination 1
  • Chest radiograph 1
  • Thyroid studies and liver transaminase levels 1
  • Digoxin level, prothrombin time, and INR when appropriate 1
  • Pulmonary function tests, including diffusing capacity 1

Regular Monitoring

  • Every six months: thyroid studies and liver transaminase levels 1
  • Digoxin level as appropriate 1
  • History and physical examination directed at detecting adverse effects 1
  • For suspected pulmonary toxicity: chest radiograph and pulmonary function tests 1
  • For visual symptoms: ophthalmologic examination 1

Drug Interactions

  • Amiodarone inhibits metabolism of several drugs through cytochrome P450 pathways 1
  • Close monitoring of digoxin levels and prothrombin time in patients taking these medications 1
  • Effects of interaction with warfarin and digoxin peak seven weeks after initiation 1
  • Grapefruit juice can inhibit amiodarone metabolism and lead to elevated drug levels 1

Practical Considerations

  • Keep amiodarone dosage at the lowest effective level to minimize side effects 1
  • The long half-life (average 58 days) means adverse effects may persist after discontinuation 1
  • Due to high lipid solubility, amiodarone is stored in fat, muscle, liver, lungs, and skin 1
  • Almost all patients develop some side effects at maintenance doses of 600 mg daily 3

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Amiodarone-Induced Thyroid Dysfunction: A Clinical Update.

Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association, 2018

Research

Toxic and therapeutic effects of amiodarone in the treatment of cardiac arrhythmias.

Journal of the American College of Cardiology, 1983

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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