Can patients with normal insulin levels taking SGLT2 (Sodium-Glucose Linked Transporter 2) inhibitors develop euglycemic diabetic ketoacidosis (DKA)?

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Euglycemic DKA in Patients Taking SGLT2 Inhibitors with Normal Insulin Levels

Yes, patients with normal insulin levels taking SGLT2 inhibitors can develop euglycemic diabetic ketoacidosis (DKA). 1 This risk exists even in individuals without diabetes mellitus who have sufficient baseline insulin levels.

Mechanism and Risk

  • SGLT2 inhibitors lower glucose concentrations, altering the insulin/glucagon ratio and predisposing patients to ketosis, regardless of their baseline insulin status 1
  • When physiological stress occurs, counterregulatory hormones drive hyperketonaemia, potentially leading to euglycemic DKA 1
  • Until recently, it was believed that people without diabetes had sufficient insulin to prevent significant ketosis, but recent publications have challenged this assumption 1
  • Several case reports document euglycemic DKA in patients without diabetes mellitus who were taking SGLT2 inhibitors 1

Diagnostic Challenges

  • Euglycemic DKA presents with normal or minimally elevated glucose levels (<200 mg/dL or <11.0 mmol/L), making diagnosis difficult 1, 2
  • The condition is characterized by high anion gap metabolic acidosis, elevated serum and urine ketones, with serum glucose <250 mg/dL 3
  • The absence of hallmark symptoms like hyperglycemia, polyuria, and polydipsia often leads to delayed diagnosis 4, 5

Precipitating Factors

  • Reduced oral intake or fasting 1, 4
  • Major acute illness 4
  • Surgery or invasive procedures 1, 6
  • Insulin reduction or omission 1, 6
  • Dehydration 1
  • Excessive exercise 6
  • Low-carbohydrate diets 6
  • Excessive alcohol intake 1, 6
  • Chronic pancreatitis 4

Incidence and Risk Comparison

  • The risk of perioperative DKA is higher in patients taking SGLT2 inhibitors compared to those not taking them (1.02 vs. 0.69 per 1000 patients) 1
  • In non-operative settings, DKA incidence is greater in patients with diabetes mellitus than those without (1 in 339 vs. 1 in 15,592) 1
  • Emergency surgery carries a higher risk of DKA than elective surgery (1.1% vs. 0.17%) 1

Prevention Strategies

  • Patients should be informed about the risk of euglycemic DKA and advised to seek immediate care if symptoms develop (nausea, vomiting, abdominal pain, generalized weakness) 1
  • Avoid substantial initial reductions in insulin dose (>20%) after initiation of SGLT2 inhibitors 1
  • For perioperative management, SGLT2 inhibitors should be omitted the day before and the day of a procedure 1
  • Maintain adequate hydration and avoid prolonged fasting periods 1
  • Monitor glucose and ketone levels during high-risk periods 1
  • Consider glucose-containing intravenous fluids during unavoidable prolonged fasting 1

Management of Suspected Cases

  • All SGLT2 inhibitor-treated patients presenting with signs or symptoms of DKA should be investigated for DKA, especially if euglycemic 6
  • If DKA is diagnosed, SGLT2 inhibitor treatment should be stopped immediately 6
  • Treatment includes intravenous insulin, dextrose-containing fluids, and electrolyte replacement 5

Special Considerations

  • Patients on complex insulin regimens or with a history of labile blood glucose should have SGLT2 inhibitors initiated in collaboration with diabetes care providers 1
  • Approximately 5-10% of adult-onset diabetes is late-onset type 1; these patients have an increased risk of DKA 1
  • The risk of euglycemic DKA exists as a continuum rather than having a defined threshold when it will not occur 1

In conclusion, the evidence clearly demonstrates that euglycemic DKA can occur in patients with normal insulin levels who are taking SGLT2 inhibitors. Healthcare providers should maintain a high index of suspicion for this condition in any patient on SGLT2 inhibitors presenting with symptoms suggestive of DKA, regardless of their glucose levels or diabetes status.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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