What is the incidence of Chronic Thromboembolic Pulmonary Hypertension (CTEPH) in patients with Venous Thromboembolism (VTE)?

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Last updated: October 10, 2025View editorial policy

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Incidence of Chronic Thromboembolic Pulmonary Hypertension (CTEPH) in VTE Patients

The cumulative incidence of CTEPH after symptomatic pulmonary embolism is reported to be between 0.1% and 9.1% within the first two years after the event, with more recent data suggesting approximately 2-4% of patients develop this complication. 1

Epidemiology and Incidence

  • CTEPH is a rare but serious complication of VTE, occurring in approximately 3% of patients after acute pulmonary embolism according to recent estimates 2
  • The wide range in reported incidence (0.1-9.1%) is likely due to referral bias, absence of early symptoms, and difficulty differentiating acute PE from an acute episode superimposed on pre-existing CTEPH 1
  • In a recent Japanese registry (COMMAND VTE Registry-2), the cumulative diagnosis of CTEPH after acute PE was 2.3% at 3 years in the direct oral anticoagulant era 3
  • Most cases of CTEPH develop within the first two years after the index PE event, with no patients developing CTEPH later than 2 years after the index PE in some studies 1
  • Importantly, a significant number of CTEPH cases (approximately 60%) develop in patients with no antecedent history of acute VTE 1

Risk Factors for CTEPH Development

  • Female gender (HR 2.09) 3
  • Longer interval from symptom onset to diagnosis of PE 3
  • Hypoxemia at PE diagnosis (HR 2.52) 3
  • Right heart load/dysfunction (HR 9.28) 3
  • Unprovoked PE (HR 2.77) 3
  • Other risk factors include:
    • Inadequate anticoagulation 1
    • Large thrombus mass and residual thrombi 1
    • Recurrence of VTE 1
    • Presence of lupus anticoagulant or antiphospholipid antibodies 1
    • Elevated levels of coagulation factor VIII 1
    • History of splenectomy, ventriculoatrial shunt, inflammatory bowel disease, and chronic osteomyelitis 1

Diagnostic Considerations

  • Routine screening for CTEPH after PE is not supported by current evidence 1
  • An echocardiogram performed approximately 6 weeks after acute PE might help predict subsequent CTEPH development 1
  • V/Q scintigraphy is the best screening test for CTEPH, as a normal V/Q scan effectively excludes the diagnosis 1, 2
  • A relatively normal CT angiogram can be observed in CTEPH despite substantial V/Q scan abnormalities 1
  • Definitive diagnosis requires:
    • Mean pulmonary arterial pressure ≥25 mmHg at rest
    • Pulmonary capillary wedge pressure ≤15 mmHg
    • Evidence of thrombotic pulmonary artery obstruction after at least 3 months of effective anticoagulation 1

Clinical Implications

  • CTEPH is a debilitating disease with poor prognosis if left untreated 1
  • In untreated patients, mortality rates of 70% have been reported among those with mean pulmonary artery pressure >40 mmHg, increasing to 90% at >50 mmHg 1
  • Early recognition is challenging due to the non-specific nature of symptoms and the median time from symptom onset to diagnosis is 14 months 1
  • Pulmonary thromboendarterectomy is potentially curative with mortality rates of approximately 2% at expert centers 2

Pathophysiology

  • CTEPH results from incomplete resolution of pulmonary emboli with subsequent vascular remodeling 1
  • Residual thrombus remains despite adequate anticoagulation at 1 year in as many as half of all patients 1
  • Beyond major pulmonary vascular obstruction, CTEPH also involves pulmonary microvascular disease, which may contribute to disease progression 1

Understanding the relatively low but significant incidence of CTEPH following VTE is important for clinicians to maintain appropriate vigilance, particularly in patients with risk factors or persistent dyspnea after PE.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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