What is the medical term for low bicarbonate levels?

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Medical Term for Low Bicarbonate Levels

The medical term for low bicarbonate levels in the blood is metabolic acidosis, characterized by serum bicarbonate concentrations below 22 mmol/L. 1

Understanding Metabolic Acidosis

  • Metabolic acidosis refers to a condition where there is a primary reduction in serum bicarbonate (<22 mmol/L), typically associated with blood pH <7.35 1
  • It occurs when there is a relative accumulation of plasma anions in excess of cations, which reduces plasma pH 2
  • The body attempts to compensate for metabolic acidosis by increasing ventilation to eliminate CO₂ 1

Classification of Metabolic Acidosis

Metabolic acidosis can be classified based on the serum anion gap:

  • Normal anion gap (hyperchloremic) acidosis: Often seen in renal tubular acidosis or diarrhea 2
  • High anion gap acidosis: Commonly occurs in conditions like diabetic ketoacidosis, lactic acidosis, or advanced chronic kidney disease 3
  • These categories can overlap in some clinical scenarios 2

Causes of Low Bicarbonate

  • Renal causes: Decreased ability to excrete acids or reabsorb bicarbonate in chronic kidney disease 4
  • Increased acid production: From conditions like diabetic ketoacidosis or lactic acidosis 2
  • Gastrointestinal losses: Bicarbonate loss through diarrhea 5
  • Diet-related: Western diets high in animal protein and low in fruits and vegetables can lead to chronic low-grade metabolic acidosis 6

Clinical Significance

  • Low bicarbonate levels are associated with muscle wasting, bone disease, hypoalbuminemia, and inflammation 4
  • In chronic kidney disease, metabolic acidosis can accelerate kidney function decline through multiple pathways 6
  • Acidosis stimulates endothelin 1 secretion and angiotensin II production, which may promote renal fibrosis 6
  • Correction of acidemia has been associated with increased serum albumin and decreased protein degradation rates 6

Diagnostic Considerations

  • A comprehensive evaluation should include measurement of serum bicarbonate, arterial blood gases (to determine pH and PaCO₂), and calculation of the anion gap 1
  • It's important to differentiate between true metabolic acidosis and hypobicarbonatemia due to chronic respiratory alkalosis, which can be misdiagnosed 7
  • Urine anion gap can be used as a surrogate marker of urine ammonium excretion to help differentiate between metabolic acidosis and chronic respiratory alkalosis 7

Management Implications

  • Treatment should focus on addressing the underlying cause of metabolic acidosis 8
  • In chronic kidney disease, bicarbonate supplementation is suggested when serum bicarbonate concentration is <22 mmol/L 1, 4
  • Sodium bicarbonate supplementation or increased fruit and vegetable intake can help treat high net endogenous acid production 6
  • Increasing fruit and vegetable intake not only raises plasma bicarbonate levels but may also decrease systolic blood pressure and body weight 6

Monitoring Recommendations

  • In patients with chronic kidney disease, serum bicarbonate should be measured monthly 6
  • The goal is to maintain serum bicarbonate at or above 22 mmol/L to prevent complications of metabolic acidosis 6, 1

References

Guideline

Acid-Base Disorders and Bicarbonate Levels

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Metabolic Acidosis of CKD: An Update.

American journal of kidney diseases : the official journal of the National Kidney Foundation, 2016

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Metabolic Acidosis or Respiratory Alkalosis? Evaluation of a Low Plasma Bicarbonate Using the Urine Anion Gap.

American journal of kidney diseases : the official journal of the National Kidney Foundation, 2017

Guideline

Acidosis Treatment Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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