What is Conus Medullaris Syndrome (CMS)?

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Conus Medullaris Syndrome (CMS)

Conus medullaris syndrome (CMS) is a neurological disorder resulting from injury to the conus medullaris, the terminal portion of the spinal cord, characterized by varying degrees of lower limb weakness, sensory disturbances, and bowel/bladder dysfunction.

Anatomy and Pathophysiology

  • The conus medullaris is located at the end of the spinal cord and continues to the cauda equina, typically ending at the L1-L2 disc space by 2 months after birth 1
  • Radiographically, a conus medullaris that ends below the middle third of the L2 vertebra is considered tethered 1
  • CMS results from damage to the conus medullaris, which can be caused by various factors including trauma, disc herniation, tumors, or inflammatory conditions 2, 3
  • Unlike cauda equina syndrome (CES), which is a pure lower motor neuron injury, CMS often presents with a mixed upper motor neuron and lower motor neuron syndrome 4

Clinical Manifestations

  • Symptoms of CMS are age-dependent and may include pain, sensorimotor disturbances of the lower limbs, and difficulties with bladder and/or bowel control 1
  • Back and/or leg pain is common and may be dull and aching, sharp, lancinating, electrical, or dysesthetic in character 1
  • Pain may be aggravated by flexion and extension of the spine or by walking or running 1
  • Sensory abnormalities generally start distally in the leg and become more proximal over time; occasionally a "suspended" sensory loss may be present 1
  • Bladder and bowel dysfunction may manifest as urinary and fecal urgency and/or incontinence, urinary tract infections, dribbling urinary stream, incomplete emptying, or inability to void 1
  • Perineal or saddle anesthesia is a characteristic finding 2, 3
  • In some cases, CMS may present without lower extremity involvement, which is extremely rare 2

Diagnostic Evaluation

  • MRI is the preferred imaging method for diagnosing CMS as it provides better visualization of soft tissue, bone marrow, and spinal canal 5
  • CT can be used as a supplementary imaging method, especially when evaluating bony structures 5
  • Diagnosis should be based on a combination of clinical examination and imaging diagnostics 5
  • Post-myelographic CT may be useful in cases where direct compression of the conus medullaris is suspected 2

Differential Diagnosis

  • Cauda equina syndrome (CES) - a pure lower motor neuron injury affecting the nerve roots below the conus medullaris 3, 4
  • Tethered cord syndrome - results from physical stretching of the spinal cord leading to impaired blood flow and metabolic failure 1
  • Inflammatory conditions such as MOG-associated disease can present with conus medullaris syndrome 6
  • High-riding conus medullaris syndrome - presents with more exclusive upper motor neuron signs 7

Management

  • Early diagnosis and treatment are crucial to avoid long-term neurological deficits 5
  • Surgical decompression is often necessary for traumatic or compressive lesions of the conus medullaris 2, 3
  • Posterior decompression and stabilization offers at least equivalent neurological outcomes as nonoperative or anterior approaches for traumatic injuries 4
  • For inflammatory causes like MOG-associated disease, corticosteroids may be effective 6
  • Early transfer to specialized spinal cord injury centers is crucial but often delayed, potentially depriving patients of timely interventions 8

Prognosis

  • The prognosis for CMS varies depending on the cause, timing of intervention, and extent of injury 3, 4
  • Patients with CMS often retain ambulatory motor function but may have persistent bowel, bladder, and sexual dysfunctions 4
  • Surgical treatment for CMS without lower extremity involvement caused by intradural disc herniation has shown effectiveness 2
  • High-riding injuries to the conus medullaris may have a more prolonged recovery period due to upper motor neuron involvement 7

Complications and Long-term Outcomes

  • Long-standing untreated tethering can result in progressive musculoskeletal deformities and/or scoliosis 1
  • Muscle atrophy may become apparent, with thinning of calf muscles and/or "saber shins" 1
  • Orthopedic deformities of the feet and spine, such as progressive scoliosis and exaggerated lumbosacral lordosis, may develop 1
  • With long-standing tethering, the skin of the leg and foot may become thin, shiny, and hairless due to autonomic changes 1

References

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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