What gene is responsible for teicoplanin (glycopeptide antibiotic) resistance in Enterococcus Faecalis (E. Faecalis)?

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Teicoplanin Resistance in Enterococcus faecalis: Associated Genes

Teicoplanin resistance in Enterococcus faecalis is primarily associated with the vanA gene, while vanB-type E. faecalis can develop teicoplanin resistance through mutations in the vanB gene cluster. 1, 2

Glycopeptide Resistance Phenotypes in Enterococci

  • Enterococci display several phenotypes of vancomycin resistance (vanA through vanE), with vanA, vanB, and vanC being most commonly associated with infective endocarditis 1
  • The vanA phenotype is characterized by high-level vancomycin resistance (MIC ≥64 μg/mL) and teicoplanin resistance 1
  • The vanB phenotype typically shows intermediate to high-level vancomycin resistance (MIC 16-512 μg/mL) but initial susceptibility to teicoplanin 1, 2
  • The vanC phenotype exhibits low to intermediate-level resistance (MIC 2-32 μg/mL) 1

Genetic Basis of Teicoplanin Resistance

  • The vanA gene is primarily responsible for teicoplanin resistance in E. faecalis and is found in both E. faecium and some strains of E. faecalis 1, 2
  • VanB-type E. faecalis can develop teicoplanin resistance through mutations in the vanB gene cluster, as demonstrated in clinical isolates 2
  • In VanB-type strains that develop teicoplanin resistance, SDS-PAGE analysis has shown constitutive production of a normally inducible 41-kDa protein 2

Development of Teicoplanin Resistance

  • VanB-type E. faecalis can acquire teicoplanin resistance in vivo during treatment, challenging the utility of teicoplanin for infections caused by vancomycin-resistant enterococci 2
  • In vitro studies show that teicoplanin can select for teicoplanin-resistant mutants at frequencies of 10^-6 to 10^-7 3
  • Two-step acquisition of resistance to teicoplanin-gentamicin combinations has been observed in experimental models, requiring mutations affecting both antibiotics 3

Clinical Implications

  • Susceptibility testing should include penicillin, vancomycin (MIC determination), and testing for high-level resistance to aminoglycosides 1
  • For strains resistant to β-lactams, vancomycin, or aminoglycosides, in vitro susceptibility to daptomycin and linezolid should be obtained 1
  • Vancomycin-resistant E. faecalis strains are less common (approximately 3%) compared to vancomycin-resistant E. faecium 4
  • For multidrug-resistant enterococcal infections, consultation with infectious disease specialists is recommended 1

Treatment Considerations

  • Combination therapy is often required for enterococcal infections with glycopeptide resistance 1, 5
  • Synergistic bactericidal activity has been demonstrated with double β-lactam combinations (imipenem and ampicillin or cephalosporins plus ampicillin) for some resistant E. faecalis strains 1
  • Despite low-level VanB-type resistance, combination therapy with a glycopeptide and aminoglycoside (if no high-level aminoglycoside resistance) can provide effective bactericidal activity 5

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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