What is the mechanism of action (MOA) of sucralfate?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: October 21, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Mechanism of Action of Sucralfate

Sucralfate exerts its therapeutic effect primarily through formation of an ulcer-adherent complex that covers the ulcer site and protects it against further attack by acid, pepsin, and bile salts. 1

Primary Mechanisms

  • Sucralfate forms an ulcer-adherent complex with proteinaceous exudate at the ulcer site, creating a protective barrier against acid, pepsin, and bile salts 1, 2
  • When exposed to gastric acid, sucralfate becomes a viscous adhesive substance that selectively binds to damaged mucosa through electrostatic interactions between its negatively charged polyanions and positively charged proteins in mucosal lesions 2, 3
  • Sucralfate provides a barrier to diffusion of hydrogen ions, inhibiting their penetration into the ulcer site 1, 4
  • It inhibits pepsin activity in gastric juice by approximately 32% when given in therapeutic doses 1
  • Sucralfate effectively adsorbs bile salts, further protecting the ulcer from damaging agents 1, 4

Secondary Mechanisms

  • Sucralfate enhances natural defensive mechanisms of the gastric mucosa by stimulating:
    • Mucus secretion
    • Bicarbonate release
    • Prostaglandin production
    • Mucosal cell renewal 5
  • It has a trophic effect on the entire mucosa, facilitating healing and re-epithelialization 5
  • Sucralfate provides minimal acid neutralization capacity (approximately 14-16 mEq per 1g dose) 1

Pharmacokinetic Properties

  • Sucralfate is minimally absorbed from the gastrointestinal tract (only 3-5% of an orally administered dose) 1, 4
  • The small amounts absorbed are excreted primarily in the urine 1
  • More than 90% of the dose is excreted unchanged in the feces 4
  • Sucralfate remains at the site of gastric ulcers for up to six hours 4
  • Its action is local rather than systemic, explaining its excellent safety profile and minimal side effects 1, 6

Clinical Applications

  • Sucralfate is effective for treating duodenal and gastric ulcers 4, 6
  • It can be used for stress ulcer prophylaxis in critically ill patients 5
  • Sucralfate enemas have shown efficacy in controlling bleeding from radiation proctitis by stimulating epithelial healing and forming a protective barrier 7, 8
  • When used for stress ulcer prophylaxis, sucralfate may be associated with a lower risk of ventilator-associated pneumonia compared to acid-suppressive therapies 7, 8

Important Considerations

  • Sucralfate should be administered 1 hour before meals and at bedtime for optimal effect 4
  • It should be administered at least 2 hours apart from drugs that decrease gastric acidity (PPIs or H2-blockers) to avoid interaction 9
  • The most common side effect is constipation, occurring in approximately 2-4% of patients 4, 6
  • Sucralfate can cause feeding tube blockage when administered through tubes, especially when combined with crushed tablets 9

References

Research

Mechanisms of action of sucralfate.

Journal of clinical gastroenterology, 1981

Research

The mechanism of protective, therapeutic and prophylactic actions of sucralfate.

Scandinavian journal of gastroenterology. Supplement, 1987

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Gastrointestinal Ulcer and Acid Reflux Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Sucralfate Administration Timing

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.