What is the classification, mechanism of action (MOA), and common side effects of atomoxetine (Strattera)?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: October 21, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Atomoxetine: Classification, Mechanism of Action, and Side Effects

Atomoxetine (Strattera) is a selective norepinephrine reuptake inhibitor (SNRI) that increases both noradrenaline and dopamine levels in the prefrontal cortex by binding to the norepinephrine transporter, with a distinct side effect profile including nausea, vomiting, fatigue, decreased appetite, abdominal pain, and somnolence. 1

Classification and Pharmacology

  • Atomoxetine is classified as a selective norepinephrine reuptake inhibitor (SNRI), not a stimulant, making it the first non-stimulant medication approved for ADHD treatment 1
  • Unlike stimulants, atomoxetine is not a controlled substance in the US, as it has negligible risk of abuse or misuse 2, 3
  • Atomoxetine is FDA-approved for ADHD treatment in both children/adolescents and adults 1, 4
  • It is available in capsules containing 10,18,25,40,60,80, or 100 mg of atomoxetine hydrochloride and as an oral solution (4 mg/ml) 1

Mechanism of Action

  • Atomoxetine selectively inhibits presynaptic norepinephrine transporters, increasing synaptic noradrenaline levels 1
  • In the prefrontal cortex, norepinephrine transporters also regulate dopamine reuptake (as dopamine transporters are scarce in this region) 1
  • This dual action increases both noradrenaline and dopamine concentrations in the prefrontal cortex synapses 1
  • The medication has high affinity and selectivity for norepinephrine transporters but little or no affinity for other neurotransmitter receptors 2

Pharmacokinetics

  • Atomoxetine is orally administered and can be given either as a single daily dose or split into two evenly divided doses 1, 2
  • It is primarily metabolized through the cytochrome P450 2D6 (CYP2D6) pathway 1
  • Approximately 7% of the population are poor CYP2D6 metabolizers, resulting in higher plasma levels and longer half-lives, potentially increasing adverse effects 1, 5
  • Some selective serotonin reuptake inhibitors (SSRIs) can elevate serum atomoxetine levels through CYP2D6 inhibition 1
  • Titration follows a sequential, weight-based approach with maximum recommended dosage of 1.4 mg/kg/day or 100 mg/day, whichever is lower 1

Common Side Effects

  • The most common adverse effects in children and adolescents include: 1, 5

    • Gastrointestinal effects: nausea, vomiting, abdominal pain
    • Decreased appetite
    • Fatigue and somnolence
    • Weight decreased
  • In adults, common side effects include: 5, 4

    • Dry mouth
    • Insomnia
    • Nausea
    • Decreased appetite
    • Constipation
    • Dizziness
    • Sexual dysfunction (in approximately 2% of adult patients) 6

Important Safety Concerns and Monitoring

  • Suicidal ideation: Analysis of twelve placebo-controlled trials showed increased risk of suicidal ideation in children and adolescents (but not in adults) 1

  • Cardiovascular effects: Atomoxetine can cause statistically but not clinically significant increases in heart rate and blood pressure 2

  • Growth effects: Initial loss in expected height and weight may occur but typically returns to normal in the longer term 2

  • Special warnings exist for: 1

    • Preexisting cardiovascular diseases/significant cardiac abnormalities
    • Emergent psychotic or manic symptoms
    • Bipolar disorder
    • Aggressive behavior or hostility
    • Possible allergic reactions
    • Effects on urine outflow
    • Priapism (rare)
  • Liver injury: Rare cases of serious liver injury have been reported 2

Clinical Considerations

  • Atomoxetine has a slower onset of action (6-12 weeks) compared to stimulants 1

  • It provides "around-the-clock" symptom control unlike stimulants 1

  • Atomoxetine may be particularly useful for: 3, 4

    • Patients at risk of substance abuse
    • Those with comorbid anxiety or tics
    • Patients who do not wish to take a controlled substance
  • Poor CYP2D6 metabolizers experience more adverse effects than extensive metabolizers 5

  • Children treated with atomoxetine should be monitored closely for suicidality, clinical worsening, and unusual behavior changes, especially during the first few months of treatment or at times of dose change 1

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.