What is the pathophysiology of pseudohyponatremia (false low sodium levels) in the setting of hyperglycemia (elevated blood glucose)?

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From the Research

Pathophysiology of Pseudohyponatremia

Pseudohyponatremia, or false low sodium levels, can occur in the setting of hyperglycemia (elevated blood glucose) due to several mechanisms:

  • The electrolyte exclusion effect, where elevated concentrations of serum solids (such as proteins or lipids) displace serum water, resulting in a reciprocal depression in serum sodium values 1
  • An increase in the measured sample's water concentration post-dilution, which can lower the sodium concentration in the sample 1
  • Serum hyperviscosity, which can reduce serum delivery to the device that apportions serum and diluent, leading to inaccurate sodium measurements 1

Laboratory Methods and Pseudohyponatremia

The occurrence of pseudohyponatremia is influenced by the laboratory method used to measure serum sodium levels:

  • Indirect ion-specific electrode (ISE) methods, which require pre-measurement sample dilution, can produce spuriously low sodium values in the presence of elevated serum solids 1, 2, 3
  • Direct ISE methods, which do not require sample dilution, are less susceptible to pseudohyponatremia 1, 3

Clinical Implications

Pseudohyponatremia can lead to mismanagement of sodium levels in clinical practice, highlighting the importance of understanding and differentiating it from true hyponatremia:

  • Patients with pseudohyponatremia do not develop water movement across cell membranes and clinical manifestations of hypotonic hyponatremia 1
  • Inadvertent correction treatment for pseudohyponatremia can be potentially detrimental 1
  • Careful clinical correlation is needed to avoid mismanagement of sodium levels in patients with hyperglycemia and pseudohyponatremia 4

References

Research

Pseudohyponatremia: a reappraisal.

The American journal of medicine, 1989

Research

Pseudohyponatremia.

The American journal of emergency medicine, 1985

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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