What is the mechanism of action (MOA) of Wellbutrin (bupropion)?

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Mechanism of Action of Wellbutrin (Bupropion)

Bupropion works primarily by inhibiting the reuptake of norepinephrine and dopamine neurotransmitters, without significant effects on serotonin neurotransmission. 1, 2

Primary Neurochemical Effects

  • Bupropion is a relatively weak inhibitor of neuronal uptake of norepinephrine and dopamine 1
  • Unlike many other antidepressants, bupropion does not inhibit monoamine oxidase or affect serotonin reuptake 1, 2
  • The medication activates the central nervous system to release norepinephrine, dopamine, and other neurotransmitters 3
  • Bupropion's mechanism differs from traditional antidepressants like SSRIs, which primarily affect serotonin 2, 4

Metabolic Considerations

  • Bupropion undergoes extensive metabolism in humans to form three active metabolites: hydroxybupropion, threohydrobupropion, and erythrohydrobupropion 1
  • Hydroxybupropion is formed via hydroxylation of the tert-butyl group of bupropion, primarily through the CYP2B6 enzyme 1
  • The amino-alcohol isomers threohydrobupropion and erythrohydrobupropion are formed via reduction of the carbonyl group 1
  • The metabolites, particularly hydroxybupropion, contribute to the clinical effects of bupropion as they reach plasma concentrations as high or higher than the parent drug 1, 2

Hypothalamic Effects

  • Bupropion administration leads to increased activation of anorexigenic neurons in the hypothalamus 3
  • This hypothalamic effect helps explain why bupropion is considered weight-neutral or weight-loss promoting, unlike many other antidepressants that cause weight gain 5, 6
  • Clinical trials have found that bupropion decreases body weight by suppressing appetite and reducing food cravings 5

Clinical Implications of Mechanism

  • The unique dual norepinephrine and dopamine reuptake inhibition mechanism explains bupropion's distinctive clinical profile 4
  • Unlike serotonergic antidepressants, bupropion has a lower incidence of sexual dysfunction, weight gain, and somnolence 7, 4
  • Common side effects related to its mechanism include nervousness and insomnia, reflecting its activating properties 2
  • Extended-release formulations moderate the stimulant-like effects by providing gradual drug delivery 6
  • The mechanism also explains bupropion's efficacy in smoking cessation, as dopaminergic effects may help reduce nicotine cravings 2, 8

Important Distinctions from Other Antidepressants

  • Bupropion is chemically unrelated to tricyclic, tetracyclic, or selective serotonin reuptake inhibitor antidepressants 1
  • Its structure closely resembles that of diethylpropion and is related to phenylethylamines 1
  • The lack of serotonergic activity distinguishes bupropion from most other modern antidepressants 2, 4
  • This unique pharmacological profile makes bupropion particularly useful for patients who experience side effects with serotonergic antidepressants 8, 4

References

Research

Bupropion: pharmacology and therapeutic applications.

Expert review of neurotherapeutics, 2006

Guideline

Mechanism of Action of Bupropion

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

A Review of the Neuropharmacology of Bupropion, a Dual Norepinephrine and Dopamine Reuptake Inhibitor.

Primary care companion to the Journal of clinical psychiatry, 2004

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Bupropion Classification and Effects

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

15 years of clinical experience with bupropion HCl: from bupropion to bupropion SR to bupropion XL.

Primary care companion to the Journal of clinical psychiatry, 2005

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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