What are the implications of an alpha-1 antitrypsin (A1AT) test result showing a phenotype of MZ?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: March 26, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the Guidelines

I strongly recommend genetic counseling and regular monitoring of lung and liver health for patients with an alpha-1 antitrypsin (A1AT) test result showing a phenotype of MZ. The MZ phenotype indicates that the patient is a carrier of one normal M allele and one deficient Z allele for alpha-1 antitrypsin, resulting in approximately 60% of normal AAT protein levels 1. This carrier state poses a slightly increased risk of developing emphysema, particularly if the patient smokes or has other environmental exposures. Key considerations for management include:

  • Complete avoidance of smoking
  • Minimization of exposure to occupational and environmental pollutants
  • Regular pulmonary function tests to monitor lung health
  • Periodic assessment of liver function due to a small increased risk of liver disease
  • Consideration of testing for family members, as this is a genetic condition
  • Early intervention with bronchodilators and standard COPD management if respiratory symptoms develop. It's essential to note that the MZ phenotype itself does not typically require augmentation therapy, which is reserved for severe deficiency states, such as the ZZ phenotype 1.

From the Research

Implications of an MZ Phenotype

An alpha-1 antitrypsin (A1AT) test result showing a phenotype of MZ has several implications:

  • The individual has an intermediate deficiency of A1AT, which may increase the risk of lung disease, particularly in smokers 2, 3.
  • The risk of emphysema is more pronounced in MZ smokers, especially when combined with other environmental challenges 2.
  • Augmentation therapy is not supported by objective studies for MZ individuals 4, 2.
  • The risk of lung disease in MZ individuals is comparable to that of SZ-AATD, but not as high as ZZ-AATD 5.
  • Some studies suggest that MZ phenotype alone may not carry a greater risk of developing lung disease than the M phenotype 3.
  • However, there have been cases reported where MZ individuals have developed lung disease, such as apical lung bullae and spontaneous pneumothorax, particularly in combination with smoking 6.

Clinical Considerations

  • Smoking cessation and elimination of other risk factors for lung disease are crucial for MZ individuals 4.
  • Careful monitoring of lung function and consideration of environmental exposures are important for MZ individuals 2.
  • The decision to prescribe augmentation therapy should be made with caution and based on individual patient factors, as there is limited evidence to support its use in MZ individuals 4, 2.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.