Can spironolactone cause hyponatremia?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: October 29, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Spironolactone Can Cause Hyponatremia

Yes, spironolactone can cause hyponatremia, particularly at higher doses or when used in combination with other diuretics. According to the FDA drug label, hyponatremia is a recognized adverse effect of spironolactone listed under electrolyte and metabolic abnormalities 1.

Mechanism and Risk Factors

  • Spironolactone is an aldosterone antagonist that acts primarily through competitive binding of receptors at the aldosterone-dependent sodium-potassium exchange site in the distal convoluted tubule 2
  • Hyponatremia occurs in 8-30% of patients treated with diuretics and is related to impaired ability of the kidneys to excrete free water 2
  • Risk factors that increase the likelihood of spironolactone-induced hyponatremia include:
    • Higher doses (50-100 mg) compared to lower doses (25 mg) 3
    • Concomitant use with other diuretics, especially furosemide 3
    • Advanced age 3
    • Diabetes mellitus 3
    • Alcohol consumption 3
    • Cirrhosis 2

Evidence from Clinical Guidelines

  • In patients with cirrhosis and ascites, hyponatremia is a common complication of diuretic therapy, with serum sodium <130 mmol/L occurring in 21.6% of patients 2
  • Hyponatremia has been associated with higher prevalence of refractory ascites, hepatic encephalopathy, spontaneous bacterial peritonitis, hepatorenal syndrome, and increased mortality 2
  • Recent guidelines define hyponatremia as serum sodium <135 mmol/L, with 130-135 mmol/L, 125-129 mmol/L, and <125 mmol/L constituting mild, moderate, and severe hyponatremia, respectively 2

Clinical Management

  • Monitor serum electrolytes periodically during spironolactone therapy 1
  • For patients with moderate hyponatremia (serum sodium 121-125 mmol/L), consider stopping diuretics and observing the patient 2
  • All experts recommend stopping diuretics if serum sodium is ≤120 mmol/L 2
  • If there is significant increase in serum creatinine or if creatinine is >150 μmol/L, volume expansion may be necessary 2
  • Fluid restriction is generally not necessary in treating most patients with cirrhosis and ascites, as chronic hyponatremia in this setting is seldom morbid 2
  • However, severe hyponatremia (serum sodium <120-125 mmol/L) may warrant fluid restriction 2

Comparison with Other Diuretics

  • A population-based case-control study found that spironolactone was associated with an increased risk of hospitalization due to hyponatremia (adjusted OR 1.96) 4
  • The risk was highest with newly initiated therapy (adjusted OR 3.55) 4
  • In contrast, furosemide showed an inverse correlation with hospitalization due to hyponatremia during ongoing use (adjusted OR 0.52), suggesting a potential protective effect 4
  • Hydrochlorothiazide can cause rapid development of hyponatremia when added to the combination of spironolactone and furosemide 2

Special Considerations

  • In heart failure patients, the risk of hyponatremia with spironolactone may be lower when used as part of optimal therapy with close monitoring by a dedicated heart failure team 5
  • The response to spironolactone varies significantly between patients, with up to sevenfold variation in potassium response, suggesting individualized dosing and monitoring is necessary 6
  • In critically ill patients receiving furosemide infusions, high-dose spironolactone (100 mg three times daily) did not significantly affect serum sodium levels compared to placebo in a small pilot study 7

Common Pitfalls and Caveats

  • Fixed-dose thiazide-spironolactone combination tablets may not reliably prevent hypokalemia due to variability in response between patients 6
  • Patients with cirrhosis and chronic hyponatremia are often asymptomatic and seldom need treatment 2
  • Attempts to rapidly correct hyponatremia in cirrhotic patients with hypertonic saline can lead to more complications than the hyponatremia itself 2
  • When hyponatremia develops in a patient on long-term furosemide therapy, causes other than the diuretic should be sought 4

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.