What causes catatonia?

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Causes of Catatonia

Catatonia is caused by a wide range of psychiatric, neurological, and medical conditions, with the most common associations being mood disorders, psychotic disorders, and various medical conditions that affect central nervous system function. 1, 2

Primary Psychiatric Causes

  • Mood Disorders: Catatonia frequently occurs in patients with primary mood disorders, including depression and bipolar disorder, and is recognized as an episode specifier in these conditions 3
  • Schizophrenia and Psychotic Disorders: Historically associated with schizophrenia (coded as F20.2 - Catatonic schizophrenia), catatonia can occur as a subtype of schizophrenia and other psychotic disorders including schizoaffective disorder, schizophreniform disorder, and brief psychotic disorder 4, 3
  • Autism Spectrum Disorders: Catatonic symptoms can present in individuals with autism spectrum disorders 5

Medical and Neurological Causes

  • Autoimmune Disorders: Anti-NMDA receptor encephalitis is a prominent cause of catatonia, with glutamatergic activity playing a key role in the pathophysiology 5, 2
  • Metabolic Disturbances: Conditions such as hyponatremia can induce catatonic states 5
  • Neurological Conditions: Cerebral venous sinus thrombosis and other neurological disorders can present with catatonia 5
  • Infectious Processes: Neuroinfections, including viral encephalitis and meningitis, can manifest with catatonic features 6
  • Neoplastic Conditions: Neoplastic meningoencephalitis and other brain tumors can present with catatonia and akinetic mutism 6

Medication-Related Causes

  • Medication Withdrawal: Abrupt discontinuation of benzodiazepines or clozapine can precipitate catatonia, likely due to sudden changes in GABA activity 5
  • Neuroleptic Malignant Syndrome: A severe reaction to antipsychotic medications that can present with catatonic features 5

Pathophysiological Mechanisms

  • Neurotransmitter Imbalance: Evidence points to an imbalance in neurotransmission involving GABA, dopamine, and glutamate systems 2

  • Neuroanatomical Pathways: Dysfunction in cortico-cortical and cortico-subcortical pathways appears central to catatonia, involving structures such as 2:

    • Orbitofrontal and temporal cortex
    • Basal ganglia
    • Brainstem structures involved in motor processing
  • Genetic Factors: Research has identified genetic factors related to oligodendrocyte function that may contribute to catatonia susceptibility 2

Developmental and Environmental Factors

  • Prenatal Viral Exposure: There is evidence suggesting that prenatal exposure to viral infections may increase risk for later development of psychotic disorders with catatonic features 1

  • Pandysmaturation: Infants genetically at risk for schizophrenia may display a neurointegrative defect described as pandysmaturation, which could be a developmental precursor to later catatonic presentations 1

Clinical Subtypes

  • Inhibited (Retarded) Catatonia: Characterized by stupor, mutism, negativism, and waxy flexibility 2

  • Excited Catatonia: Characterized by psychomotor agitation, combativeness, and autonomic instability 2

  • Malignant Catatonia: A life-threatening form with hyperthermia, autonomic instability, and altered consciousness 7

Understanding the diverse causes of catatonia is essential for proper diagnosis and treatment, as early intervention is associated with better outcomes 5. The recognition that catatonia is not merely a subtype of schizophrenia but a syndrome that can occur across multiple conditions has important implications for clinical practice 3.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Catatonia in DSM-5.

Schizophrenia research, 2013

Guideline

Catatonia Classification and Coding

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Catatonia Update.

The primary care companion for CNS disorders, 2017

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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