Posterior Tibial Tendon Dysfunction: Clinical Presentation and Differentiation from Inflammatory Arthritis
When posterior tibial tendon dysfunction (PTTD) presents with pain, swelling, and erythema that mimics inflammatory arthritis, the key distinguishing features are localized medial foot tenderness along the tendon course, inability to perform single-leg heel raise, and the "too many toes" sign—rather than the diffuse joint involvement and systemic features of inflammatory arthritis. 1
Key Clinical Distinctions
PTTD Presentation
- Localized medial foot pain and swelling along the posterior tibial tendon course from behind the medial malleolus to the navicular insertion 1, 2
- Progressive flatfoot deformity with loss of the medial longitudinal arch 3, 1
- "Too many toes" sign visible when viewing the foot from behind, indicating hindfoot valgus 1
- Inability to perform single-leg heel raise test—a pathognomonic finding 1, 4
- Insidious onset with load-related pain that worsens with activity 5
- Absence of joint effusions, which are uncommon in tendinopathy but suggest intra-articular pathology 5
Inflammatory Arthritis Features That Differ
- Multiple joint involvement rather than isolated tendon pathology 5
- Morning stiffness lasting >30 minutes (less prominent in PTTD) 5
- Systemic symptoms and elevated inflammatory markers in active inflammatory disease 5
- Enthesitis at multiple sites in spondyloarthropathies, detectable by ultrasound 5
Critical Diagnostic Pitfall
The presence of multiple symptomatic tendons should alert you to consider rheumatic disease or prompt rheumatologic referral 5. However, PTTD can occur secondary to inflammatory arthritis—particularly rheumatoid arthritis—where 11-64% of patients may develop posterior tibial tendon dysfunction depending on diagnostic criteria used 4. In rheumatoid arthritis, there is complex interplay between hindfoot joint disruption from inflammation and secondary tendon dysfunction 4.
Diagnostic Approach
Physical Examination Specifics
- Palpate the entire posterior tibial tendon course for well-localized tenderness from medial malleolus to navicular 5, 1
- Assess for muscle atrophy in the calf, indicating chronicity 5
- Perform single-leg heel raise test—inability indicates significant dysfunction 1, 4
- Observe foot alignment for pes planus deformity and "too many toes" sign 3, 1
- Check for joint effusions—their presence suggests inflammatory arthropathy rather than isolated tendinopathy 5
Imaging Strategy
- Ultrasound is the initial imaging modality of choice when performed by experienced operators, showing 100% sensitivity and 93% accuracy for posterior tibial tendon pathology 5
- Ultrasound can also detect inflammatory features including intratendinous tophi in gout, enthesitis in spondyloarthritis, and tenosynovitis in rheumatoid arthritis 5
- MRI is more sensitive than ultrasound for posterior tibial tendon pathology (>90% sensitivity), though this difference doesn't significantly affect clinical management 5
- Plain radiographs assess for flatfoot deformity, joint space narrowing, and erosions suggesting inflammatory arthritis 1, 2
Management When Inflammatory Features Present
Initial Conservative Treatment (First 6-8 Weeks)
- Relative rest with activity modification to reduce repetitive tendon loading 5
- Foot orthoses or customized arch supports to support the medial longitudinal arch 5, 6
- NSAIDs for short-term pain relief (effective for acute symptoms but no long-term benefit) 5
- Cryotherapy with melting ice water through wet towel for 10-minute periods 5
- Immobilization with CAM walker or cast in acute or refractory cases 5
Critical Corticosteroid Caveat
Avoid intratendinous corticosteroid injections as they may reduce tensile strength and predispose to spontaneous rupture 5. Peritendinous injections should be used with caution, and corticosteroid injections are specifically not recommended for Achilles-related pathology 5.
Escalation Criteria
- Refer to podiatric foot and ankle surgeon if no improvement after 6 weeks of conservative management 5, 6
- Failure to manage symptoms conservatively is the most frequent reason for surgical referral (86.2% of cases) 6
- Fixed deformity warrants surgical evaluation 6
If Inflammatory Arthritis Confirmed
- Co-manage with rheumatology for systemic disease control 5, 4
- Treatment of primary hindfoot instability from inflammatory arthritis is similar to primary PTTD, making the distinction less critical clinically 4
- Continue tendon-specific conservative measures while addressing underlying inflammatory disease 4