What are the causes of transaminitis?

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Causes of Transaminitis

Most Common Causes

Non-alcoholic fatty liver disease (NAFLD) is the most common cause of mild transaminitis in developed countries, followed by alcoholic liver disease, drug-induced liver injury, and viral hepatitis. 1, 2, 3

Primary Etiologies (in order of prevalence):

  • NAFLD/NASH: The leading cause of asymptomatic transaminase elevation, strongly associated with metabolic syndrome components including obesity, type 2 diabetes, hypertension, and hypercholesterolemia 4, 1, 3

  • Alcoholic liver disease: Second most common cause; requires detailed quantification of alcohol consumption including duration and amount 1, 2

  • Drug-induced liver injury (DILI): Common hepatotoxic medications include methotrexate, NSAIDs, statins, anticonvulsants, antiarrhythmics, tamoxifen, nitrofurantoin, minocycline, and infliximab 4, 1, 5

  • Viral hepatitis: Hepatitis B and C are frequent causes requiring screening with hepatitis B surface antigen and hepatitis C antibody 1, 3

Uncommon but Important Causes

  • Autoimmune hepatitis: Presents with persistently elevated transaminases, hyperglobulinemia, and positive autoantibodies (ANA, anti-smooth muscle antibody); affects women 3-4 times more than men 4, 1

  • Hereditary hemochromatosis: Identified through iron studies (elevated ferritin and transferrin saturation) 1, 3

  • Celiac disease: Associated with transaminase elevations that improve or normalize with gluten-free diet in 75-100% of cases 4

Rare Causes

  • Wilson disease: Copper metabolism disorder requiring ceruloplasmin testing 2, 3

  • Alpha-1 antitrypsin deficiency: Hereditary condition causing liver disease 2, 3

Context-Specific Causes

In Cancer Patients:

  • Immune checkpoint inhibitor-induced liver injury (ILICI): Immune-mediated hepatotoxicity from ICIs, distinct from traditional DILI 4
  • Primary liver tumors or hepatic metastases 4
  • Biliary obstruction (intra- or extrahepatic) 4
  • Systemic infection or sepsis 4

In HIV-Infected Patients:

  • Coinfection with hepatitis B or C viruses 6
  • Antiretroviral therapy hepatotoxicity 6
  • HIV itself causing direct liver damage 6
  • NAFLD from metabolic comorbidities 6

In Rheumatoid Arthritis Patients on Methotrexate:

  • NASH-like pattern is the most prevalent histological finding in patients with persistent transaminitis during low-dose MTX treatment 5
  • Risk factors mirror those of NAFLD: obesity (OR 3.23), type 2 diabetes (OR 3.52), hypercholesterolemia (OR 2.56), and hyperuricemia (OR 3.52) 5

Extrahepatic Causes

Important pitfall: Not all transaminase elevations originate from the liver. 3

  • Muscle disorders: Particularly affect AST levels (myopathies, rhabdomyolysis, vigorous exercise) 4, 3
  • Hemolysis: Releases AST from red blood cells 2, 3
  • Thyroid disorders: Both hypo- and hyperthyroidism can elevate transaminases 3
  • Celiac disease: Causes transaminitis independent of liver pathology 4, 3
  • Cardiac conditions: Congestive heart failure causes hepatic congestion 4

Critical Clinical Pearls

  • Mild elevations (>1x to <3x ULN) without bilirubin elevation are often non-specific and may be related to NAFLD, dietary changes, or vigorous exercise rather than clinically significant liver injury 4

  • Statin-induced transaminitis (>3x ULN) is infrequent, often resolves with dose reduction or alternative statins, and statins are not contraindicated in chronic stable liver disease like NAFLD 4

  • Acute presentations: Approximately 40% of autoimmune hepatitis cases present as acute hepatitis with jaundice and markedly elevated transaminases (several thousand), and 30% have cirrhosis at presentation 4

  • Medication discrepancies: Over 50% of patients with liver disease have discrepancies between reported and documented medications, particularly those taking more than five medications 1

References

Guideline

Management of Transaminitis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

[Not Available].

Therapeutische Umschau. Revue therapeutique, 2013

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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