What is the mechanism of action of the drug of choice for absence seizures, specifically for a patient diagnosed with this condition?

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Drug of Choice for Absence Seizures: Mechanism of Action

The correct answer is d) Inhibition of the T-type calcium channels—this is the mechanism of action of ethosuximide, the drug of choice for childhood absence seizures.

Ethosuximide as First-Line Treatment

Ethosuximide is the drug of choice for monotherapy of childhood absence epilepsy (petit mal seizures), controlling approximately 70% of absence seizures. 1, 2

  • The drug specifically targets the pathophysiology of absence seizures by suppressing the paroxysmal three cycle per second spike and wave activity associated with lapses of consciousness 1
  • It reduces the frequency of epileptiform attacks by depressing the motor cortex and elevating the threshold of the central nervous system to convulsive stimuli 1

Mechanism of Action: T-Type Calcium Channel Blockade

Ethosuximide primarily works by reducing low threshold T-type Ca²⁺ currents in thalamic neurons, which are critical for generating the abnormal thalamocortical rhythms seen in absence seizures. 3, 4

  • The drug specifically targets T-type calcium channels in thalamocortical circuitry, which are involved in generating spike-and-wave discharges characteristic of absence seizures 3
  • Research demonstrates that T-type calcium channel blockers potently suppress absence seizures by 85-90% through attenuation of burst firing in thalamic reticular nucleus neurons 5
  • Ethosuximide also decreases persistent Na⁺ and Ca²⁺-activated K⁺ currents in thalamic and cortical pyramidal neurons 3

Additional Mechanisms

Beyond T-type calcium channel inhibition, ethosuximide has secondary effects:

  • It increases GABA release in cortical networks, elevating the ratio of network inhibition to excitation 6
  • The drug reduces cortical GABA levels in genetic absence epilepsy models and decreases elevated glutamate levels in the primary motor cortex 3

Why Other Options Are Incorrect

Option a) Sodium and calcium channel blockade describes drugs like valproate or lamotrigine, which are broader-spectrum agents 2

Option b) GABA uptake inhibition describes the mechanism of tiagabine, not ethosuximide 7

Option c) GABA potentiation describes benzodiazepines or barbiturates, which are not first-line for absence seizures 7

Clinical Considerations

  • Ethosuximide has a narrow therapeutic profile and is specifically indicated for absence seizures 3
  • It is unsuitable as monotherapy if other generalized seizures (tonic-clonic or myoclonic) coexist, as it only controls absence seizures 2
  • Valproic acid may be preferred when multiple seizure types are present, as it controls absences (75%), generalized tonic-clonic seizures (70%), and myoclonic jerks (75%) 2
  • Common side effects are dose-dependent and involve the gastrointestinal tract and central nervous system 3

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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