What increases sex hormone-binding globulin (SHBG) levels?

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Factors That Increase Sex Hormone-Binding Globulin (SHBG)

SHBG levels increase with aging, hyperthyroidism, hepatic disease, anticonvulsant medications (particularly enzyme-inducing agents), estrogen therapy, thyroid hormone supplementation, smoking, and HIV/AIDS. 1

Medication-Induced SHBG Elevation

Anticonvulsant Drugs

  • Enzyme-inducing anticonvulsants are the most clinically significant medication class that raises SHBG levels. 2, 3
  • Male epileptic patients on chronic anticonvulsant therapy demonstrate elevated SHBG concentrations alongside increased LH and FSH levels, indicating compensatory pituitary response to reduced free testosterone. 2
  • The mechanism involves hepatic enzyme induction that stimulates SHBG synthesis in the liver. 3
  • Both male and female patients on anticonvulsants show significantly elevated SHBG, which can lead to functional hypogonadism despite normal or even elevated total testosterone levels. 3

Hormonal Medications

  • Estrogen therapy directly increases SHBG production. 1, 4
  • Thyroid hormone supplementation elevates SHBG levels. 1

Endocrine Disorders

Hyperthyroidism

  • Thyroid excess is a well-established cause of elevated SHBG. 1
  • This represents a reversible cause that should be identified and treated to normalize SHBG levels. 1

Functional Hypothalamic Amenorrhea (FHA)

  • Women with FHA without polycystic ovarian morphology demonstrate higher SHBG levels associated with correspondingly higher FSH levels. 1
  • This pattern contrasts with FHA patients who have polycystic ovarian morphology, who show lower SHBG and lower FSH. 1

Hepatic Disease

  • Chronic liver disease and cirrhosis paradoxically increase SHBG despite overall hepatic dysfunction. 1
  • Liver disease disrupts the hypothalamic-pituitary axis, leading to altered gonadotropin levels including FSH elevation. 1
  • Evaluating liver function tests is essential when investigating unexplained SHBG elevation. 1

Lifestyle and Environmental Factors

Smoking

  • Cigarette smoking is associated with elevated SHBG levels. 1

Aging

  • SHBG naturally increases with advancing age in both sexes. 1
  • This age-related rise contributes to the decline in bioavailable testosterone seen in older men despite stable total testosterone. 1

Infectious Disease

HIV/AIDS

  • HIV infection and AIDS are associated with elevated SHBG levels. 1
  • HIV testing should be considered in appropriate clinical contexts when evaluating unexplained SHBG elevation. 1

Clinical Pitfalls to Avoid

  • Do not rely solely on total testosterone measurements when SHBG is elevated, as this misses functional hypogonadism. 1, 5
  • Always measure or calculate free testosterone (using total testosterone/SHBG ratio) when SHBG abnormalities are suspected. 5
  • A free testosterone index <0.3 indicates hypogonadism regardless of total testosterone levels. 5
  • Patients on anticonvulsants may have reduced sexual activity and symptoms of hypogonadism despite normal or elevated total testosterone due to SHBG-mediated reduction in free testosterone. 2
  • Consider medication review as a reversible cause before pursuing other interventions. 1

References

Guideline

The Relationship Between SHBG, Free Testosterone, and Pituitary Response

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Sex hormone changes in male epileptics.

Clinical endocrinology, 1980

Research

Effect of anticonvulsants on plasma testosterone and sex hormone binding globulin levels.

Journal of neurology, neurosurgery, and psychiatry, 1978

Research

Steroidal and non-steroidal factors in plasma sex hormone binding globulin regulation.

The Journal of steroid biochemistry and molecular biology, 1992

Guideline

Management of High Sex Hormone-Binding Globulin (SHBG) and Low Free Testosterone

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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