What are the key respiratory system concepts to know for Step 1, including anatomy, physiology, and common diseases like Chronic Obstructive Pulmonary Disease (COPD) and asthma?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: November 6, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Key Respiratory System Concepts for USMLE Step 1

COPD: Core Pathophysiology and Clinical Features

COPD is defined as a preventable and treatable disease characterized by airflow limitation that is not fully reversible, caused by chronic inflammatory response to noxious particles or gases. 1

Essential Pathophysiologic Mechanisms

  • Tobacco smoke is the primary risk factor worldwide, though biomass fuels and α1-antitrypsin deficiency are increasingly recognized 1
  • Chronic inflammation involves oxidative stress, proteinase-antiproteinase imbalance, abnormal immune responses, and accelerated lung aging 1
  • Four anatomic compartments are affected: central airways, peripheral airways, lung parenchyma, and pulmonary vasculature 1
  • Pathological hallmarks include emphysema (permanent destructive enlargement of airspaces distal to terminal bronchioles), mucus hypersecretion, ciliary dysfunction, and airflow limitation 1

Emphysema Subtypes (Critical for Step 1)

  • Centriacinar emphysema: Destroys respiratory bronchioles, alveolar ducts, and central acinar alveoli; associated with more small airways disease and less elastic recoil loss 1
  • Panacinar emphysema: Destroys the entire acinus 1
  • Paraseptal emphysema: Occurs near connective tissue septae, predisposes to pneumothorax and bullae formation 1

Clinical Presentation and Severity Classification

  • Mild COPD: FEV1 60-79% predicted with FEV1/FVC <70%; often presymptomatic 1
  • Moderate COPD: FEV1 40-59% predicted; patients present with intermittent chest problems and work difficulty 1
  • Severe COPD: FEV1 <40% predicted; significant symptoms with intermittent hospitalizations 1

Physical Examination Findings

  • Early disease: Normal examination is common 1
  • Progressive disease: Quiet breath sounds, prolonged expiratory duration, weight loss, and signs of hyperinflation 1

Asthma vs. COPD: Distinguishing Features

Airway Remodeling Differences (High-Yield)

Asthma features:

  • Reticular basement membrane thickening 2
  • Eosinophilic infiltration with CD4+ T-helper lymphocytes 2
  • Bronchial hyperreactivity as a characteristic feature 1

COPD features:

  • Epithelial squamous metaplasia and airway wall fibrosis 2
  • BAL neutrophilia with CD8+ T-cytotoxic lymphocyte infiltration 2
  • Emphysematous changes 2

Asthma-COPD Overlap (ACO)

  • ACO represents overlapping clinical phenotypes with worse outcomes than either disease alone 3, 2
  • Patients demonstrate features of both diseases with intermediate pathophysiology 2
  • No universally accepted definition exists, making diagnosis challenging 2

Respiratory Mechanics: Fundamental Concepts

Key Mechanical Properties

Three fundamental properties determine ventilation mechanics:

  • Airway resistance (Raw): Opposition to airflow through conducting airways 4
  • Lung compliance (CL): Distensibility of lung tissue 4
  • Chest wall compliance (CCW): Distensibility of thoracic cage 4

Pressure Relationships

  • Ventilation depends on pressure gradients at the airway opening, alveolar space, pleural space, and across respiratory muscles 4
  • These relationships govern both spontaneous breathing and mechanical ventilation 4

Exercise Limitation in Obstructive Disease

Peripheral Muscle Dysfunction

  • Skeletal muscle alterations render muscles susceptible to contractile fatigue, particularly during cycling 1
  • Increased lactic acid production occurs at lower work rates, increasing ventilatory requirements 1
  • Lactic acidosis is exacerbated by CO2 retention, further increasing ventilatory burden 1

Respiratory Muscle Dysfunction

  • Diaphragm adaptation: Greater fatigue resistance but mechanical disadvantage from hyperinflation 1
  • Static and dynamic hyperinflation places respiratory muscles at suboptimal length-tension relationships 1
  • Functional inspiratory muscle weakness contributes to hypercapnia, dyspnea, and reduced exercise performance 1

Comorbidities in COPD (Frequently Tested)

Cardiovascular Associations

  • Exacerbations increase cardiovascular event risk 1
  • Overlap syndrome (COPD + sleep apnea) requires CPAP treatment, which decreases mortality 1
  • Pulmonary hypertension develops in advanced disease with persistent hypoxemia 1

Systemic Effects

  • Limb muscle weakness occurs as a systemic manifestation 1
  • Chronic multimorbidity is the rule rather than exception, particularly in elderly patients 1

Patient-Centered Outcomes (Emerging Concept)

Traditional physiological outcomes (FEV1, 6-minute walk distance) correlate poorly with patient-important outcomes. 1

Critical Distinction

  • Surrogate outcomes: Physiological/anatomical measures (FEV1, imaging findings) 1
  • Patient-centered outcomes: Dyspnea, quality of life, exacerbation frequency, hospitalizations, mortality 1
  • Interventions improving surrogate outcomes frequently fail to improve patient-centered outcomes 1

Bronchodilator Therapy: FDA-Approved Use

Albuterol (Short-Acting Beta-Agonist)

  • Duration of action: 4-6 hours 5
  • Critical counseling point: Do not increase dose or frequency without physician consultation 5
  • Warning sign: When treatment becomes less effective or symptoms worsen, seek immediate medical attention 5
  • Paradoxical bronchospasm: Can occur; discontinue immediately if it develops 5
  • Common adverse effects: Palpitations, chest pain, tachycardia, tremor, nervousness 5

Pulmonary Rehabilitation Principles

Exercise Training Framework

High-intensity endurance training (>60% maximal work rate) for 20-60 minutes, 3-5 times weekly maximizes physiologic benefits. 1

  • Target intensity: Borg dyspnea/fatigue score 4-6 or RPE 12-14 1
  • Walking vs. cycling: Walking is functional and improves walking capacity; cycling loads quadriceps more specifically and causes less desaturation 1
  • Alternative for severe limitation: Low-intensity or interval training when high-intensity cannot be achieved 1

Physical Activity Monitoring

  • Physical inactivity is common in COPD and associated with poor outcomes independent of lung function 1
  • Direct measurement using accelerometry provides objective assessment of daily activity 1
  • Walking time correlates poorly with degree of airflow limitation 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Pathogenesis, clinical features of asthma COPD overlap, and therapeutic modalities.

American journal of physiology. Lung cellular and molecular physiology, 2022

Research

Phenotyping Asthma and/or COPD Using 129Xe MRI and Comprehensive Physiologic Testing.

American journal of respiratory and critical care medicine, 2025

Research

Ventilation Mechanics.

Seminars in respiratory and critical care medicine, 2023

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.