What are the characteristics and effects of pulmonary embolism (PE)?

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Pulmonary Embolism: True/False Analysis

Statement 1: Usually associated with thrombosis in deep veins of legs or pelvic veins

TRUE - Deep vein thrombosis and pulmonary embolism are clinical manifestations of the same entity, venous thromboembolic disease 1. The vast majority of pulmonary emboli originate from thrombi in the deep veins of the lower extremities or pelvic veins 2. The European Society of Cardiology guidelines explicitly state that DVT and PE are common causes of illness and death, representing different presentations of the same underlying thrombotic process 1.

Statement 2: May cause sudden death

TRUE - Pulmonary embolism can be immediately fatal and is a well-established cause of sudden death 1. The European Society of Cardiology reports that in approximately 25% of patients, the first manifestation of PE is sudden-unexpected death 3. Mortality in untreated PE is approximately 30%, and the ICOPER study revealed a cumulative 3-month mortality as high as 17.5% 1. A forensic autopsy study found that PE represented 4.3% of all sudden deaths, with 78% of cases occurring at home or during transfer to healthcare centers 3. Right ventricular failure due to acute pressure overload is the primary cause of death in severe PE 1.

Statement 3: Often results in pulmonary infarction

FALSE - Pulmonary infarction is actually uncommon in pulmonary embolism 1. The European Society of Cardiology guidelines explicitly state that "even if they do not affect haemodynamics, small distal emboli may create areas of alveolar haemorrhage resulting in haemoptysis, pleuritis, and pleural effusion, which is usually mild. This clinical presentation is known as 'pulmonary infarction'" 1. However, a forensic study found pulmonary infarction was associated with PE in only 6% of cases 3. The effect on gas exchange from pulmonary infarction is normally mild, except in patients with pre-existing cardiorespiratory disease 1.

Statement 4: Is a cause of pulmonary hypertension

TRUE - Pulmonary embolism is definitively a cause of pulmonary hypertension, both acutely and chronically 1. The European Society of Cardiology states that "in the long-term, there is the risk of developing pulmonary hypertension from recurrent embolism or the absence of reperfusion of the pulmonary vasculature" 1. Chronic thromboembolic pulmonary hypertension develops in approximately 3.8% of patients within 2 years after an initial PE episode 4. Acutely, pulmonary artery pressure increases when >30-50% of the total cross-sectional area of the pulmonary arterial bed is occluded by thromboemboli 1, 5.

Statement 5: Produces strain on the left side of the breast

FALSE - Pulmonary embolism produces strain on the RIGHT side of the heart, not the left 1. The pathophysiology clearly demonstrates that acute PE causes right ventricular failure due to acute pressure overload 1. The abrupt increase in pulmonary vascular resistance results in RV dilation and dysfunction 1, 5. While the interventricular septum bows leftward and impedes LV filling secondarily, the primary hemodynamic strain is on the right ventricle 1, 5. The European Society of Cardiology emphasizes that "acute RV failure is a critical determinant of clinical severity and outcome in acute PE" 1.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Blood Pressure in Pulmonary Embolism

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Pathophysiology of Pulmonary Thromboembolism and Obstructive Shock

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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