Evaluation and Management of Electrical Pains Over Left Breast and Chest
Any patient presenting with electrical pains over the left breast and chest requires immediate ECG within 10 minutes and urgent cardiac evaluation to exclude acute coronary syndrome (ACS), even though the "electrical" quality makes cardiac ischemia less likely. 1
Immediate Initial Actions
Obtain an ECG within 10 minutes of presentation, regardless of setting. 1 If you are in an office setting without ECG capability, immediately transfer the patient to the emergency department by EMS—do not delay for any reason. 1
If clinical evidence suggests ACS or other life-threatening causes, transport urgently to the ED by EMS, not by personal vehicle. 1 EMS provides critical advantages including prehospital ECG acquisition, trained personnel for arrhythmia management, and shorter transport times. 1
Measure cardiac troponin as soon as possible after ED arrival. 1 High-sensitivity troponin is preferred because it enables more rapid detection or exclusion of myocardial injury. 1
Obtain a chest radiograph to evaluate for cardiac, pulmonary, and thoracic causes. 1
Critical Diagnostic Considerations
Characteristics That Make Cardiac Ischemia LESS Likely
The "electrical" quality of chest pain has specific features that argue against angina, though cardiac causes must still be excluded with objective testing: 1, 2
- Sharp, stabbing, or electrical pain that increases with inspiration makes ischemic heart disease less likely (but does not rule it out). 1, 2, 3
- Pain lasting less than 5 seconds or more than 30 minutes without other symptoms is typically nonanginal. 3
- Pain reproducible with palpation or single movements of the trunk/arm suggests musculoskeletal origin. 3
- Pain localized with one finger or in an inframammary location is usually nonanginal. 3
Life-Threatening Differential Diagnoses to Exclude
Despite the atypical "electrical" quality, you must systematically rule out these conditions:
Acute Coronary Syndrome: 13% of ACS patients present with pleuritic or atypical pain. 2 Serial ECGs should be performed if initial ECG is nondiagnostic and symptoms persist. 1
Pericarditis: Sharp, pleuritic pain that worsens supine and improves sitting forward, with friction rub and widespread ST-elevation with PR depression on ECG. 1, 2, 4
Pulmonary Embolism: Presents with dyspnea, pleuritic chest pain, tachycardia (>90% of cases), and tachypnea. 2, 4 Must be excluded before considering benign etiologies. 2
Pneumothorax: Classic triad of dyspnea, pleuritic pain on inspiration, and unilateral absence of breath sounds with hyperresonant percussion. 2, 4
Aortic Dissection: Sudden onset "ripping" pain radiating to back, with pulse differential in 30% of cases. 1, 2
Myocarditis: Fever, chest pain, heart failure signs, and S3 gallop. 1, 2
Common Non-Life-Threatening Causes
After excluding emergent conditions:
Costochondritis/Tietze syndrome: Tenderness of costochondral joints on palpation. 1, 2 Critical pitfall: 7% of patients with reproducible chest wall tenderness still have ACS. 2
Herpes zoster: Pain in dermatomal distribution triggered by touch, with characteristic unilateral dermatomal rash. 1, 2
Pneumonia: Localized pleuritic pain, fever, productive cough, regional dullness to percussion, egophony. 1, 2, 4
ECG-Directed Management Algorithm
Follow this systematic approach based on ECG findings: 1
- STEMI pattern: Follow STEMI guidelines immediately. 1
- ST depression or new T-wave inversions: Follow NSTE-ACS guidelines. 1
- Diffuse ST-elevation with PR depression: Manage as pericarditis. 1
- New arrhythmia: Follow arrhythmia-specific guidelines. 1
- Nondiagnostic or normal ECG: Repeat ECG if symptoms persist or change, or if troponins become positive. 1 Consider supplemental leads V7-V9 if posterior MI suspected. 1
Critical Pitfalls to Avoid
Never assume reproducible chest wall tenderness excludes serious pathology—7% of patients with palpable tenderness have ACS. 2
Do not use nitroglycerin response as a diagnostic criterion—relief with nitroglycerin does not confirm or exclude myocardial ischemia, and cervical root compression pain and esophageal spasm can both be relieved by nitroglycerin. 5, 3
Do not delay transfer for troponin testing in office settings—patients with suspected ACS should be transported urgently to the ED by EMS without waiting for additional testing. 1, 2
Sharp, pleuritic, or "electrical" pain does not exclude cardiac ischemia—objective testing is mandatory. 1, 2, 3
Up to 6% of patients with evolving ACS are discharged from the ED with a normal ECG—serial ECGs and troponins are essential. 1
Special Population Considerations
Women are at risk for underdiagnosis—obtain history emphasizing accompanying symptoms more common in women with ACS (nausea, fatigue, dyspnea). 1, 4
Patients ≥75 years: Consider ACS when accompanying symptoms such as shortness of breath, syncope, acute delirium, or unexplained falls are present. 1, 4