What is the role of sacubitril (Angiotensin Receptor Neprilysin Inhibitor) and valsartan (Angiotensin II Receptor Blocker) in treating heart failure with reduced ejection fraction?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: November 21, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Sacubitril/Valsartan in Heart Failure with Reduced Ejection Fraction

Primary Recommendation

Sacubitril/valsartan should replace ACE inhibitors or ARBs in patients with symptomatic HFrEF (LVEF ≤40%) who remain symptomatic despite optimal medical therapy with an ACE inhibitor/ARB, beta-blocker, and mineralocorticoid receptor antagonist, as it significantly reduces cardiovascular death and heart failure hospitalization. 1, 2, 3


Treatment Algorithm for HFrEF

The European Society of Cardiology recommends a stepwise approach: 1

  • First-line therapy: ACE inhibitor + beta-blocker 1
  • Second-line therapy: Add mineralocorticoid receptor antagonist (MRA) if patient remains symptomatic 1
  • Third-line therapy: Replace ACE inhibitor/ARB with sacubitril/valsartan if patient remains symptomatic despite optimal therapy 1, 2
  • Additional therapy: SGLT2 inhibitor (dapagliflozin or empagliflozin) to further reduce hospitalization and death risk 1

Clinical Benefits and Mechanism

Sacubitril/valsartan provides dual neurohormonal modulation by enhancing the natriuretic peptide system through neprilysin inhibition while simultaneously blocking the renin-angiotensin system via AT1 receptor antagonism. 4

Key outcomes compared to ACE inhibitors: 1, 4

  • 20% reduction in cardiovascular mortality 2
  • Significant reduction in heart failure hospitalization 1, 4
  • Improved cardiac remodeling with increased LVEF and decreased left ventricular volumes 1, 5
  • Average 5% increase in ejection fraction within 3 months of treatment 5

Real-world data demonstrates that within 4 months of treatment, patients experience symptom improvements and a significant reduction in all-cause hospitalizations (from 27.5% to 17.0%, p=0.009). 6


Dosing and Titration

Starting Dose 1, 3

  • Patients on high-dose ACE inhibitors: Start 49/51 mg twice daily 1, 3
  • Patients on low/medium-dose ACE inhibitors or ARBs: Start 24/26 mg twice daily 1, 3
  • Treatment-naïve patients (de novo): Start 24/26 mg twice daily 1, 3
  • Special populations (severe renal impairment, moderate hepatic impairment Child-Pugh B, or age ≥75 years): Start 24/26 mg twice daily 1, 3

Titration Schedule 1, 3

Double the dose every 2-4 weeks as tolerated to reach the target dose of 97/103 mg twice daily, which provides maximum mortality benefit. 1, 3 Real-world data shows only 17% of patients achieve target dose after 4 months, indicating significant under-titration in practice. 6


Critical Precautions Before Initiation

Mandatory Washout Period 1, 3

A 36-hour washout period is absolutely required when switching from an ACE inhibitor to sacubitril/valsartan to avoid angioedema. 1, 3 No washout period is needed when switching from an ARB. 1

Volume Status 2

Ensure patients are not volume-depleted at initiation to avoid hypotension. 2 In non-congested patients with borderline blood pressure, consider reducing loop diuretic doses to mitigate hypotensive effects. 1, 2

Hemodynamic Stabilization for Hospitalized Patients 1

Resolution of acute pulmonary congestion is required before initiating sacubitril/valsartan in hospitalized patients. 1 Approximately 25% of patients may develop hypotension when initiated in the hospital setting. 2


Managing Hypotension

Hypotension is the most common side effect, occurring in 16.0% (asymptomatic) and 11.1% (symptomatic) of patients. 2 However, efficacy and safety are maintained despite hypotension, with benefits consistent across all baseline systolic blood pressure categories, including <110 mmHg. 2

Management strategies: 1, 2

  • For asymptomatic hypotension: Continue therapy without dose reduction 1
  • For symptomatic hypotension: Consider temporary dose reduction rather than permanent discontinuation 1
  • 40% of patients requiring temporary dose reduction can subsequently be restored to target doses 1
  • Reduce diuretic doses in non-congested patients 1, 2

Common Pitfalls to Avoid

The European Society of Cardiology specifically warns against: 1

  • Failure to titrate to target doses due to asymptomatic hypotension or mild laboratory changes 1
  • Permanent dose reductions when temporary reductions with subsequent re-titration would be more appropriate 1
  • Believing medium-range doses provide most benefits when target doses are necessary for maximum mortality benefit 1
  • Treating heart failure less aggressively than other life-threatening conditions despite similar mortality risks 1

Monitoring Requirements

Close follow-up with serial assessments after initiation: 2

  • Blood pressure monitoring, especially during initiation and titration 1, 2
  • Electrolytes, particularly when used with aldosterone antagonists 1
  • Renal function 1, 2

Drug Interactions

Sacubitril/valsartan increases levels of statins that are substrates of OATP1B1, OATP1B3, OAT1, and OAT3 transporters. 1 Consider lower doses of atorvastatin, fluvastatin, pitavastatin, pravastatin, rosuvastatin, or simvastatin when used in combination. 1


Emerging Evidence in Other Populations

Recent pooled analysis of PARAGLIDE-HF and PARAGON-HF demonstrates that sacubitril/valsartan reduces cardiovascular and renal events in heart failure with mildly reduced ejection fraction (HFmrEF, LVEF 41-49%) and preserved ejection fraction (HFpEF), particularly in those with LVEF ≤60%. 1, 7, 8 Treatment benefits were larger in patients with LVEF ≤60% (rate ratio 0.78) compared to those with LVEF >60% (rate ratio 1.09). 8


Contraindications

  • Concomitant use with ACE inhibitors 3
  • History of angioedema related to previous ACE inhibitor or ARB therapy requires caution 1
  • Pregnancy (drugs acting on the renin-angiotensin system cause fetal injury and death) 3

References

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.